Reference Protocols

Peptide Protocols

Five sections — community biohacking stacks, physician-supervised clinic protocols, fitness community protocols, cross-community consensus findings, and highly experimental frontier protocols. All include documented risks and mitigation where data exists.

Section One
Biohacking Community
7 protocols · research compounds · self-directed
Section Two
Peptide Clinic
8 protocols · physician supervised · compoundable
Section Three
Cross-Community Consensus
6 findings · both worlds independently agree
Section Four
Fitness Community
4 protocols · strongman · bodybuilding · aesthetics · endurance
Clinic — 08
Age-Related Decline
Full replacement protocol · 40+ · hormonal + GH + NAD+ + mito
⚡ Section Five
Highly Experimental
6 protocols · frontier · animal data + community n=1

Important: All protocols on this page are for educational and informational purposes only. Biohacking protocols involve research compounds not approved for human use. Clinic protocols require physician supervision. This page does not constitute medical advice. Always consult a qualified healthcare professional before starting any peptide protocol. Documented overdose risks reflect available evidence and are not exhaustive.

Section One

Biohacking Community Protocols

Research compounds · self-directed use · community-validated over years of shared experience

Bio — 01
The Wolverine Stack
BPC-157 · TB-500 · MGF/PEG-MGF · Injury Recovery
The most searched peptide protocol online. Three complementary healing mechanisms: BPC-157 drives angiogenesis and growth factor expression at the injury site; TB-500 regulates actin dynamics and systemic tissue repair; MGF/PEG-MGF activates local satellite cells for muscle and connective tissue rebuilding. Together they cover every phase of tissue repair.
CommunityRisk: Low–Moderate
Stack — Compounds & Doses
BPC-157
250–500mcg SubQ or IM · once daily · adjacent to injury site
Stable acetate form. Can also be taken orally (500mcg–1mg) for gut healing specifically. Inject as close to target tissue as practically possible.
→ Entry page
TB-500
2–2.5mg SubQ · twice weekly (loading) → once weekly (maintenance)
Loading phase: 2–2.5mg 2x/week for 4–6 weeks. Maintenance: 2mg once weekly. Systemic distribution means site injection less critical than BPC-157.
→ Entry page
PEG-MGF
200mcg SubQ · twice weekly · optional addition
Add to stack for significant muscle or connective tissue injury. Activates satellite cells via MAPK-Erk1/2 — non-redundant with BPC-157 and TB-500. Most useful for tears and partial ruptures.
→ Entry page
Protocol & Timeline
Week 1–2Acute phase. BPC-157 daily + TB-500 loading (2–2.5mg twice weekly). Pain and swelling typically begin reducing. Angiogenesis signalling initiated.
Week 3–6Active repair. Continue same protocol. Most users report 40–60% pain reduction and improved mobility by week 4. Add PEG-MGF if not already running — satellite cell activation peaks during this phase.
Week 6–10Consolidation. Drop TB-500 to maintenance (once weekly). BPC-157 can continue daily or drop to 5 days on/2 off. Continue until full function restored.
CyclingRun 8–12 weeks on, 4–8 weeks off. For chronic conditions some users run indefinitely at maintenance doses — safety data does not prohibit this but long-term human data is absent.
⚠ Documented Risks & Overdose Concerns
low Injection site reactions — Redness, minor swelling at injection site. Rotate sites. Self-resolving within 24–48 hours.
low BPC-157 proliferative risk — Theoretical concern at very high doses over extended periods — BPC-157 has no established toxicity ceiling in animal models but promotes growth factor expression.
moderate MGF/PEG-MGF proliferative concern — Prolonged supraphysiological satellite cell activation raises theoretical proliferative risk. Limit PEG-MGF to 4–6 week cycles. Contraindicated with active or recent malignancy.
low No known acute overdose — BPC-157 and TB-500 have wide therapeutic windows. No documented acute toxicity from dose escalation in human use. PEG-MGF — keep below 400mcg per dose.
🔬 Blood Work · Injury Recovery
Before StartingBaseline
General Health Panel Once
  • Full blood count
  • Metabolic panel — kidney and liver function
  • hsCRP — baseline inflammation
During ProtocolWeeks 4, 8, 12
Inflammation Tracking Every 4 weeks
  • hsCRP — should trend down
  • Pain score (VAS 0–10) — self-reported weekly
  • Range of motion — physiotherapist measured monthly
Post-Protocol4 weeks after
Healing Assessment Once
  • hsCRP — confirm return to baseline
  • Imaging if available
  • Functional movement assessment
♀ Female note: No significant dose differences for BPC-157, TB-500, or PEG-MGF. Women often report slightly faster soft tissue healing response. Same blood work protocol applies.
Bio — 02
GH Optimisation Stack
Ipamorelin/CJC-1295 · MK-677 · IGF-1 LR3 (Advanced)
The layered growth hormone stack — from conservative (secretagogue only) through intermediate (add MK-677) to advanced (add IGF-1 LR3). Each layer adds potency and complexity. Most community users run the first two layers; IGF-1 LR3 is for experienced users only due to hypoglycaemia risk.
CommunityRisk: Moderate–High (IGF-1 LR3)
Stack — Compounds & Doses
Ipamorelin / CJC-1295
200mcg ipamorelin + 100mcg CJC-1295 (no DAC) · SubQ · 2–3x daily
Inject fasted (minimum 2h post-meal). Pre-sleep injection mandatory — amplifies natural GH pulse during slow-wave sleep. Pre-workout and morning are optional additions.
→ Entry page
MK-677 (Ibutamoren)
10–25mg oral · once nightly at bedtime
Layer 2 — adds 24-hour GH elevation on top of pulsatile secretagogue injections. Start at 10mg to assess hunger and water retention before escalating. Bedtime dosing maximises sleep benefit and minimises daytime hunger.
→ Entry page
IGF-1 LR3 (Advanced only)
20–50mcg SubQ · post-workout · experienced users only
Layer 3. Inject immediately post-training. ALWAYS have fast-acting carbohydrates available. Start at 20mcg and monitor glucose response before escalating. Do not combine with insulin without CGM.
→ Entry page
Protocol & Timeline

Layer 1 (Conservative): Ipamorelin/CJC only. 8–12 week cycles, 4 weeks off. Most clinically supported. Monitor IGF-1 at baseline and 8 weeks.

Layer 2 (Intermediate): Add MK-677 nightly. Run alongside Ipamorelin/CJC or standalone. MK-677 can run longer term — monitor fasting glucose and HbA1c every 3 months. Add berberine 500mg 2x/day if glucose trends up.

Layer 3 (Advanced): Add IGF-1 LR3 post-workout on training days only. Maximum 4–6 week cycles with equal off time. Monitor for hypoglycaemia symptoms — sweating, confusion, tremor. If any symptoms: eat fast carbs immediately and reduce dose 50%.

Synergy: Ipamorelin/CJC drives pulsatile GH pulses; MK-677 maintains elevated 24h GH/IGF-1 baseline; IGF-1 LR3 delivers direct systemic IGF-1R activation for 20–30 hours per dose. Together these approach — but do not equal — the total GH axis effect of pharmaceutical HGH.

⚠ Documented Risks & Overdose Concerns
moderate IGF-1 LR3 hypoglycaemia — Documented and acute. Blood glucose can drop dangerously, especially fasted or overnight. Always administer with or immediately after food. Keep fast carbs accessible.
moderate MK-677 insulin resistance — Fasting glucose increases ~5mg/dL average at 25mg/day. Dose-dependent. Monitor HbA1c quarterly. Contraindicated in T2D without physician oversight.
low Cortisol/prolactin (if GHRPs added) — Ipamorelin is highly selective — minimal HPA activation. GHRP-2 or GHRP-6 substitution raises cortisol and prolactin more significantly.
moderate Water retention / joint ache — Common with MK-677 at 25mg, especially weeks 1–3. Usually self-resolving. Dose reduction to 10–15mg typically resolves without losing sleep benefit.
serious IGF-1 LR3 + insulin combination — Absolutely do not combine without continuous glucose monitoring (CGM). Additive hypoglycaemia risk is potentially lethal. CGM is non-negotiable if both are used.
low Long-term IGF-1 elevation concerns — Chronically elevated IGF-1 epidemiologically associated with increased cancer incidence. Keep IGF-1 LR3 cycles short. Annual IGF-1 blood level monitoring recommended.
🔬 Blood Work · GH Optimisation Stack
Before StartingEssential baseline
Metabolic Panel Once
  • IGF-1 — non-negotiable baseline
  • Fasting glucose + insulin + HbA1c
  • Full metabolic panel
Hormonal Panel Once
  • Full hormonal panel
  • Thyroid (TSH)
  • Cortisol (if adding GHRPs)
During ProtocolRegular monitoring
Core Metabolic Monthly
  • IGF-1 — target mid-normal; above 2× ULN = reduce dose
  • Fasting glucose — watch for upward trend with MK-677
  • HbA1c — quarterly at MK-677 above 15mg
IGF-1 LR3 Layer Only Weekly first month
  • Post-dose glucose 1h after injection
  • Symptom check: sweating, confusion, tremor = hypoglycaemia
  • Fasting glucose morning of injection days
Post-Cycle4–6 weeks after
Recovery Panel Once
  • IGF-1 — confirm baseline return
  • Fasting glucose
  • Prolactin + cortisol if GHRPs used
♀ Female note: Women reach equivalent IGF-1 at 30–50% lower doses. IGF-1 LR3: start at 10–20mcg. MK-677: 5–10mg often sufficient. Female IGF-1 target: 120–230 ng/mL.
Bio — 03
HGH Biohacking Protocol
Pharmaceutical Human Growth Hormone · Community Doses & Approach
Biohackers use pharmaceutical-grade HGH (Somatropin) — typically sourced from grey-market pharmacies — at doses well above clinical replacement but below the bodybuilding performance-enhancement range. The goal is body composition optimisation, anti-ageing effects, and recovery enhancement. Carries significant risks at higher doses that must be taken seriously.
Research / Grey MarketRisk: High at Performance Doses
Stack — Compounds & Doses
Somatropin (Pharmaceutical HGH)
1–3 IU SubQ · anti-ageing / body composition range
Anti-ageing/longevity community typically uses 1–2 IU/day. Performance-oriented biohackers use 2–4 IU/day. Above 4 IU/day enters bodybuilding territory with substantially elevated risk. Inject SubQ in abdomen, alternating sites.
→ Entry page
Ipamorelin/CJC-1295 (support)
200mcg/100mcg · 2x daily · optional alongside HGH
Some biohackers add secretagogues to preserve pituitary function and maintain some pulsatility alongside exogenous HGH. Logic: HGH suppresses endogenous GH — secretagogues may limit this suppression, though evidence is limited.
→ Entry page
Berberine or Metformin
500mg 2–3x/day with meals
Essential insulin sensitivity management. HGH at doses above 2 IU/day reliably induces insulin resistance. Berberine is community standard; metformin requires prescription. Monitor fasting glucose monthly.
Protocol & Timeline
Week 1–4Start at 1 IU/day. Assess: water retention (common, resolves weeks 3–4), joint ache, sleep quality improvement, morning energy. Monitor fasting glucose.
Week 4–8If 1 IU well tolerated, optionally increase to 1.5–2 IU. Body composition changes become visible — waist reduction, muscle fullness. Skin quality improvement often noted.
Month 3–6Maintenance phase at chosen dose. IGF-1 blood test to confirm levels are within 1.5–2× upper limit of normal range. Above 2× ULN — reduce dose. Annual CT abdomen if using above 2 IU long-term.
CyclingCommunity split: some run 6 months on/2 months off; others run year-round at low dose (1 IU). No consensus. Exogenous HGH suppresses pituitary GH production during use — off-cycles allow recovery.
⚠ Documented Risks & Overdose Concerns
moderate Carpal tunnel syndrome — Dose-dependent. Tingling and numbness in hands — often the first warning sign of too-high dose. Reduce dose by 50%, reassess. Usually resolves within weeks of dose reduction.
moderate Insulin resistance / glucose elevation — HGH is diabetogenic at supraphysiological doses. Fasting glucose elevation at 2+ IU/day is near-universal without insulin sensitiser. Monitor monthly; add berberine/metformin if trending up.
moderate Water retention and oedema — Particularly weeks 1–4. Sodium and fluid accumulation in soft tissues. Usually self-resolving; dose reduction helps. Raises blood pressure — monitor if history of hypertension.
serious Acromegaly at chronic high doses — At doses above 4–6 IU/day for extended periods: irreversible jaw, hand and organ growth. The hands, nose and jaw enlarge first. If any extremity growth noticed — stop immediately. This is an early reversibility window.
serious IGF-1 mediated tumour promotion — Supraphysiological IGF-1 stimulates cellular proliferation broadly. If undiagnosed cancer present, HGH may promote growth. Annual health screening is non-negotiable at any dose above 1 IU/day.
critical Never use insulin alongside HGH without expert guidance — The combination used by bodybuilders for maximum anabolism carries genuine risk of fatal hypoglycaemia. CGM mandatory if both are used. Outside the scope of biohacking — this is competitive bodybuilding territory.
🔬 Blood Work · HGH Biohacking Protocol
Before StartingNon-negotiable baseline
Metabolic & Safety Once
  • IGF-1 — establish true baseline
  • Fasting glucose + HbA1c + fasting insulin
  • Full liver panel
  • Kidney function (eGFR, creatinine)
  • Full blood count + haematocrit
Cardiovascular Once
  • Lipid panel
  • Blood pressure
  • Resting ECG if over 40 or family cardiac history
Hormonal Once
  • Full hormonal panel
  • Thyroid
  • PSA (men over 40)
During ProtocolRegular monitoring
IGF-1 & Glucose Monthly
  • IGF-1 — target mid-normal; above 2× ULN = reduce dose immediately
  • Fasting glucose — should not rise above 100 mg/dL
  • HbA1c — quarterly
  • Blood pressure — monthly
Safety Markers Every 6–8 weeks
  • Liver enzymes
  • Haematocrit — quarterly
  • Hand and jaw self-assessment — early acromegaly detection
Post-Cycle / AnnualAfter stopping + yearly
Recovery & Screening Annual
  • IGF-1 6 weeks post-cycle
  • Full metabolic panel 8 weeks post
  • Comprehensive health + cancer screen annually
  • Bone density (DEXA) annually at doses above 2 IU/day
♀ Female note: Women require 0.2–1 IU/day vs 1–4 IU/day in men. Female IGF-1 target: 120–230 ng/mL. Monthly bloods for first 6 months. Mammography annually.
Bio — 04
Cognitive Enhancement Stack
NA Semax Amidate · Selank · Cerebrolysin · Cycling Protocol
The biohacking cognitive stack built on BDNF upregulation, anxiolytic modulation, and neurotrophic support. Designed for sustained cognitive enhancement rather than stimulant-like acute effect — changes build over weeks as BDNF drives structural changes in dendritic architecture. Selank prevents the anxiety some users experience from Semax alone.
CommunityRisk: Low–Moderate
Stack — Compounds & Doses
N-Acetyl Semax Amidate
200–400mcg intranasal · morning · 5 days on / 2 days off
Start at 200mcg — 2–4× more potent per mcg than base Semax. One puff per nostril from a 0.1% solution. Tolerance builds with continuous daily use — cycle 5/2 or take week breaks monthly.
→ Entry page
Selank
250mcg intranasal · as needed or daily
Anxiolytic companion — prevents overstimulation from Semax alone. Can be taken separately (morning calm, Semax focus) or simultaneously. Community often uses Selank intranasal 30 min before Semax.
→ Entry page
Cerebrolysin
5–10mL IM or IV · 10-day course · 2–3x/year
The heavy hitter — multiple neurotrophic factors, 10-day intensive courses rather than daily dosing. IM is community-accessible; IV is more effective but requires clinic or experienced practice. BDNF, NGF, CNTF in one preparation.
→ Entry page
Protocol & Timeline
Week 1–2Semax + Selank daily (5/2 cycle). Acute effects: clarity, focus, reduced anxiety. Most noticeable in the first hour after intranasal administration. Some users report mild headache first 2–3 days — reduce dose.
Week 3–6Cumulative BDNF effects build. Working memory and information processing improvements reported consistently by week 4. Sleep quality often improves alongside cognitive effects.
Month 2–3Schedule a Cerebrolysin 10-day course — IM injection 5–10mL daily for 10 consecutive days. Many users report this as the most significant cognitive upgrade in the protocol. Continue Semax/Selank cycle around it.
OngoingCerebrolysin 2–3x/year. Semax/Selank 5/2 cycling indefinitely or with monthly week-long breaks. Some users rotate Semax and NA Semax Amidate across cycles to prevent tolerance.
⚠ Documented Risks & Overdose Concerns
low Semax overstimulation — At doses above 600mcg: anxiety, irritability, mental overactivation. Reduce dose. Selank co-administration prevents this in most users.
moderate Cerebrolysin quality/sourcing — Genuine Cerebrolysin (Ebewe Pharma, Austria) must be refrigerated. Counterfeit products are common. Purchase from verified pharmacy suppliers only. Improper storage destroys activity.
low Semax tolerance with continuous daily use — Daily use beyond 2–3 weeks produces diminishing returns. The 5/2 cycle prevents this. Effect typically fully restored after 48-hour break.
moderate Cerebrolysin contraindications — Contraindicated in epilepsy, severe renal impairment, and with concurrent MAOI antidepressants. Check medication interactions before use.
🔬 Blood Work · Cognitive Stack
Before StartingBaseline
Baseline Cognitive Panel Once
  • Thyroid (TSH, T3, T4) — thyroid dysfunction mimics cognitive issues
  • Full blood count — anaemia impairs cognition
  • Vitamin B12 + folate
  • Fasting glucose — insulin resistance impairs cognition
  • hsCRP — neuroinflammation baseline
During ProtocolEvery 8–12 weeks
Monitoring Every 8–12 weeks
  • Thyroid — Semax can affect thyroid axis
  • Standardised cognitive test (Cambridge Brain Sciences or similar)
  • Sleep quality scores
Post-ProtocolOptional
Assessment Once
  • Repeat cognitive benchmarks vs baseline
  • Mood + anxiety scales (PHQ-9, GAD-7)
  • Thyroid if new symptoms
♀ Female note: No significant dose differences. Some women cycle Semax with menstrual cycle — pausing luteal phase can prevent agitation. Thyroid monitoring especially important for women.
Bio — 05
Longevity Protocol
Epitalon · NAD+/NMN · SS-31 · GHK-Cu · MOTS-c
The hallmarks-of-ageing stack — addressing telomere length (Epitalon), NAD+ decline (NMN/NR/5-Amino-1MQ), mitochondrial function (SS-31, MOTS-c), and tissue maintenance (GHK-Cu). Each compound targets a different ageing mechanism. Best run as a sustained lifestyle protocol rather than acute cycles.
CommunityRisk: Low
Stack — Compounds & Doses
Epitalon
5–10mg SubQ or IV · 10-day course · 2x/year
Pineal gland tetrapeptide — upregulates telomerase, regulates melatonin production, anti-tumour activity in animal models. Run intensive 10-day courses twice yearly. Some community members run shorter monthly maintenance doses.
→ Entry page
NMN or NR + 5-Amino-1MQ
NMN 500–1000mg oral daily + 5-Amino-1MQ 50mg daily
Complementary NAD+ approach: NMN/NR adds precursors (supply side); 5-Amino-1MQ blocks NNMT to reduce NAD+ catabolism (consumption side). Together more effective than either alone.
→ Entry page
SS-31 (Elamipretide)
2–4mg SubQ · daily or 5x/week
Mitochondrial inner membrane peptide — FDA-approved for Barth syndrome (SS-31 = Bendavia). The most evidence-backed mitochondria-targeted peptide available. Daily or 5x/week SubQ injection.
→ Entry page
GHK-Cu
Topical 1–2% daily + 0.5–2mg SubQ 2–3x/week
Copper peptide — drives collagen synthesis, activates over 4,000 genes, anti-inflammatory. Topical for skin; SubQ for systemic gene regulation and tissue maintenance effects.
→ Entry page
MOTS-c
5–10mg SubQ · 3–5x/week
Mitochondria-encoded metabolic peptide — AMPK activation, insulin sensitisation, exercise-like metabolic effects. Elevated in centenarians. Run continuously or in 4-week cycles with 2 weeks off.
→ Entry page
Protocol & Timeline

Morning routine: NMN/NR + 5-Amino-1MQ oral with breakfast. SS-31 SubQ. MOTS-c SubQ.

Evening/night: GHK-Cu topical to face/hands. SubQ GHK-Cu 2–3x/week (separate from topical application days).

Twice yearly: Epitalon 10-day intensive course (10mg/day SubQ or IV). Many users align these with seasonal transitions — spring and autumn.

Tracking: This protocol is designed for long-term assessment — short-term subjective effects are subtle. Track biological age markers annually: telomere length testing (available commercially), epigenetic clocks (TruAge, Levine), comprehensive blood panel including IGF-1, inflammatory markers (CRP, IL-6), and metabolic markers.

⚠ Documented Risks & Overdose Concerns
low Epitalon telomerase activation — Theoretical concern: telomerase activation in cancer cells. In practice, Epitalon has anti-tumour activity in animal models. Contraindicated with active malignancy as a precaution.
low SS-31 injection site reactions — Minor injection site redness. Most common adverse effect. Self-resolving. Rotate sites.
low NMN/NR and flushing — Some users experience flushing (more common with NR at high doses). Reduce dose or switch form. Not harmful.
low 5-Amino-1MQ long-term unknown — No long-term human safety data. Off-target NNMT inhibition in non-adipose tissues not characterised. Most compelling safety concern is what we do not know.
🔬 Blood Work · Longevity Protocol
Before StartingBiological age baseline
Longevity Biomarkers Once
  • IGF-1
  • Fasting glucose + HbA1c + fasting insulin
  • Inflammatory panel: hsCRP, IL-6, TNF-α
  • Lipid panel (advanced: apoB, Lp(a))
  • Full blood count
Biological Age Tests Baseline then annually
  • Epigenetic clock (TruAge, Levine) — primary outcome
  • Telomere length
  • DHEA-S
  • NAD+ levels if available
  • Thyroid full panel
During ProtocolQuarterly
Core Metabolic Every 3 months
  • Fasting glucose — should improve
  • hsCRP — should trend down
  • IGF-1 — Epitalon can influence GH axis
  • Energy + sleep quality (weekly subjective)
Annual AssessmentOnce per year
Biological Age Retesting Annually
  • Repeat epigenetic clock
  • Telomere length comparison
  • Full comprehensive metabolic panel
  • Cardiac assessment
  • DEXA bone density
♀ Female note: No significant dose differences. Women may get enhanced GHK-Cu skin response. Compare epigenetic clock results to sex-matched references. DHEA-S declines faster post-menopause — supplement only if confirmed low.
Bio — 06
TRT Companion Protocol
Gonadorelin · Kisspeptin-10 · Testosterone Axis Preservation
When exogenous testosterone suppresses the HPG axis, the hypothalamus stops pulsing GnRH, LH and FSH collapse, and testicular function declines. This protocol works upstream to maintain the axis during TRT — preserving testicular volume, fertility potential, and the physiological signalling chain that testosterone alone cannot replace.
CommunityRisk: Low–Moderate
Stack — Compounds & Doses
Gonadorelin
100–200mcg SubQ · twice daily · morning and evening
Identical to endogenous GnRH. Pulsatile dosing essential — continuous infusion paradoxically suppresses the axis. Twice daily SubQ mimics the natural ~90-min hypothalamic pulse pattern well enough to maintain pituitary responsiveness. ~$15–20/month compounded.
→ Entry page
Kisspeptin-10
50–100mcg SubQ · 2–3x weekly
Optional upstream addition — stimulates the hypothalamic neurons that release GnRH, rather than replacing GnRH directly. For men not responding adequately to gonadorelin alone. Also has direct libido effects via limbic GPR54 receptors independent of testosterone.
→ Entry page
Aromatase Inhibitor (if needed)
Anastrozole 0.25–0.5mg · 2x weekly · physician prescribed
Increased testicular stimulation from gonadorelin may increase intratesticular oestrogen via aromatisation. Monitor oestradiol at 6–8 weeks — if elevated above reference range, add low-dose AI. Physician-prescribed only.
Protocol & Timeline
Week 1–4Start gonadorelin twice daily alongside TRT. Most users notice improved testicular fullness within 2–4 weeks. LH becomes measurable on blood panel (vs undetectable on TRT alone).
Week 6–8Blood panel: LH, FSH, testosterone, oestradiol. If LH remains undetectable — consider adding Kisspeptin-10 2–3x weekly. If oestradiol elevated — discuss AI with prescribing physician.
Week 8–12If Kisspeptin-10 added: reassess LH/FSH at 4 weeks. Should see further improvement in gonadotropin signal. Subjective: libido often improves beyond what testosterone alone achieves — the direct central GPR54 effect.
OngoingContinue indefinitely while on TRT. The goal is maintenance — this is not a cycling protocol. Blood panel every 3 months: testosterone, LH, FSH, oestradiol, haematocrit, PSA (if over 45).
⚠ Documented Risks & Overdose Concerns
critical Continuous gonadorelin dosing — axis suppression — The most important risk: dosing gonadorelin continuously (e.g. via patch or pump without pulsatility) causes receptor downregulation and paradoxically LOWERS testosterone. Twice-daily SubQ injections provide adequate pulsatility. Never use a continuous infusion device without specialist guidance.
moderate Non-response to gonadorelin — Some men do not respond adequately — LH remains suppressed despite correct dosing. Individual pituitary sensitivity varies. If LH undetectable at 8 weeks, switch to HCG (requires physician prescription) for more reliable testicular stimulation.
moderate Oestrogen elevation — Increased testicular activity raises oestradiol. Monitor E2 at 6–8 weeks. Symptoms of excess oestrogen: breast tenderness, mood changes, water retention. Add aromatase inhibitor if confirmed elevated.
low Injection frequency burden — 4 injections per day (2x TRT + 2x gonadorelin) plus periodic Kisspeptin can become burdensome. Auto-inject devices reduce friction. Some patients prefer HCG 2–3x/week for simpler protocol despite higher cost.
🔬 Blood Work · TRT Companion Protocol
Before StartingHPG axis baseline
Reproductive Hormones Once — 48h post-T injection (trough)
  • Total testosterone
  • LH + FSH — establish suppression level
  • Oestradiol E2 — sensitive assay
  • SHBG
  • Prolactin
Health Baseline Once
  • Full blood count + haematocrit
  • PSA (men over 45)
  • Metabolic panel
  • Lipid panel
During ProtocolQuarterly
Reproductive Panel Every 3 months — always trough
  • Total testosterone — target 500–800 ng/dL
  • LH + FSH — should be measurable (gonadorelin working)
  • Oestradiol E2 — target 20–35 pg/mL
  • SHBG — if symptoms persist
Safety Markers Every 3 months
  • Haematocrit — must stay below 52%
  • PSA (men over 45)
  • Blood pressure
  • Lipids — annually
Fertility CheckEvery 6 months if fertility goal
Fertility Panel Every 6 months
  • Semen analysis — volume, count, motility, morphology
  • FSH — spermatogenesis activity
  • Inhibin B — Sertoli cell function
  • LH — pulsatile GnRH response
♂ Male TRT protocol — see Clinic 04b for Female TRT.
Bio — 07
Body Composition Stack
AOD-9604 · 5-Amino-1MQ · Tesamorelin · HGH Fragment 176-191
The fat loss peptide stack for biohackers — specifically targeting visceral fat, brown fat activation, and GH-mediated lipolysis without the anabolic side effects of full GH protocols. Four compounds with complementary fat-targeting mechanisms, designed to work alongside caloric deficit rather than replace it.
CommunityRisk: Low–Moderate
Stack — Compounds & Doses
AOD-9604
300mcg SubQ · fasted · once daily morning
C-terminal HGH fragment that activates beta-3 adrenergic receptors in fat cells without raising IGF-1. Lipolytic without anabolic risk. Fasted morning injection for maximum lipolytic window. Originally developed by Monash University.
→ Entry page
5-Amino-1MQ
50–75mg oral · daily with breakfast
NNMT inhibitor — raises NAD+, activates brown fat thermogenesis, reduces adipocyte size. Complementary to AOD-9604: AOD mobilises fat via adrenergic signalling; 5-Amino-1MQ activates thermogenic burning of that mobilised fat.
→ Entry page
HGH Fragment 176–191
250–500mcg SubQ · fasted · twice daily (optional)
Alternative or addition to AOD-9604. Another GH C-terminal fragment. Stronger lipolytic effect than AOD-9604 in some users, similar IGF-1 neutral profile. Use one or the other — not both — unless experienced.
→ Entry page
Tesamorelin
1–2mg SubQ · daily · FDA approved for visceral fat
The only FDA-approved fat loss peptide — approved specifically for visceral fat reduction in HIV-associated lipodystrophy. GHRH analogue that drives GH release with specific visceral fat targeting. Add for stubborn central adiposity.
→ Entry page
Protocol & Timeline
Week 1–2Start AOD-9604 (or HGH Fragment) + 5-Amino-1MQ. Assess: mild thermogenic warmth (from 5-Amino-1MQ), any injection site reactions. No dramatic immediate changes — these compounds work gradually.
Week 3–6First visible changes in body composition typically weeks 4–6 in caloric deficit. Waist measurements most reliable tracker — scale weight may not change significantly if building lean mass simultaneously.
Week 6–10Add Tesamorelin if stubborn visceral/abdominal fat is the primary concern. Visible central adiposity reduction typically confirmed by 8–10 weeks. Monitor fasting glucose monthly.
CyclingAOD-9604 and 5-Amino-1MQ: run up to 12 weeks, 4 weeks off. Tesamorelin: physician-supervised — can run 6–12 months clinically. HGH Fragment: 8–10 week cycles.
⚠ Documented Risks & Overdose Concerns
low AOD-9604 — very clean safety profile — No IGF-1 elevation, no anabolic effects, no glucose effects. The most benign compound in the stack. No serious adverse events documented at standard doses.
moderate Tesamorelin glucose elevation — Like all GHRH analogues, Tesamorelin raises GH which antagonises insulin. Monitor fasting glucose monthly. Contraindicated in active malignancy and pregnancy.
low Combined lipolysis and adrenal load — Aggressively mobilising fat stores while in caloric deficit may increase cortisol. Avoid stacking with stimulants (caffeine, ephedrine). Ensure adequate sleep.
low 5-Amino-1MQ long-term data absent — Effective and apparently well-tolerated — but no long-term human safety studies exist. Treat as a cycle compound rather than permanent daily supplement until more data available.
🔬 Blood Work · Body Composition Stack
Before StartingMetabolic baseline
Metabolic Panel Once
  • Fasting glucose + HbA1c — Tesamorelin baseline
  • IGF-1 — if adding GH-axis compounds
  • Full metabolic panel
  • Lipids
Body Composition Once
  • DEXA scan — gold standard fat mass baseline
  • Waist circumference + abdominal girth
  • Body weight
During ProtocolMonthly
Metabolic Monitoring Monthly
  • Fasting glucose — Tesamorelin GH effect
  • HbA1c — quarterly
  • Waist circumference — every 2 weeks
Progress Tracking Every 4 weeks
  • Body composition photos (consistent lighting)
  • Waist + hip measurements
  • Energy + appetite scores
Post-Protocol4–6 weeks after
Reassessment Once
  • DEXA scan — compare to baseline
  • Fasting glucose — confirm Tesamorelin effects resolved
  • HbA1c if elevated during protocol
♀ Female note: Tesamorelin: women may see stronger visceral fat response — start at 1mg/day. AOD-9604 and HGH Fragment: same doses. 5-Amino-1MQ: same dose, potentially enhanced thermogenic response in women.
Section Two

Peptide Clinic Protocols

Physician supervised · compoundable compounds · monitoring required · not DIY

Clinic — 01
Post-COVID / CIRS Protocol
Thymosin α-1 · VIP (Intranasal) · Arg-BPC-157 · Immune Reset
Used by functional medicine and CIRS-specialist physicians for post-COVID immune dysregulation and Chronic Inflammatory Response Syndrome. Thymosin α-1 resets T-regulatory balance; VIP addresses neuroinflammation and autonomic dysfunction; BPC-157 supports gut barrier repair which is central to CIRS pathology.
Physician SupervisedRisk: Low–Moderate
Stack — Compounds & Doses
Thymosin α-1
1.5mg SubQ · twice weekly · 12 weeks standard course
The most clinically validated immune peptide — 35+ country approvals, 30+ clinical trials. Restores T-regulatory function and NK cell activity. Standard CIRS protocol: 1.5mg 2x/week for 12 weeks, reassess. Some physicians extend to 6 months.
→ Entry page
VIP (Vasoactive Intestinal Peptide)
50mcg intranasal · 4x daily · titrated to tolerance
Central to Dr Shoemaker CIRS protocol. Anti-inflammatory neuropeptide targeting hypothalamic-pituitary axis dysfunction central to CIRS. Start at 50mcg once daily and titrate up over 2 weeks. Many CIRS patients experience dramatic symptom improvement.
→ Entry page
Arg-BPC-157 (oral)
500mcg–1mg oral · twice daily · with meals
Oral form preferred — gut barrier repair is central to CIRS pathology (leaky gut drives ongoing antigen exposure). Same VEGFR2 mechanism as injectable. Take 30 minutes before meals for optimal mucosal contact.
→ Entry page
Protocol & Timeline

Prerequisite: CIRS protocol should be directed by a Shoemaker-trained or CIRS-specialist physician. VIP should only be started after mould/biotoxin exposure is addressed and HERTSMI-2 score confirms safe environment.

Sequence matters: Many CIRS physicians sequence the protocol — address environmental exposure first, then use cholestyramine/binders, then start Thymosin α-1 for immune reset, then add VIP for neurological/autonomic restoration, then BPC-157 for gut repair.

Lab monitoring: CIRS requires comprehensive monitoring including TGF-β1, MMP-9, C4a, MSH, VEGF, VIP (endogenous), and osmolality. Interpret with specialist — these markers guide protocol progression.

Contraindication: VIP is contraindicated in active infection, SIBO (must be treated first), and in autoimmune conditions without specialist oversight.

⚠ Documented Risks & Overdose Concerns
low Thymosin α-1 flu-like response — Mild fatigue, low-grade fever within 24–48h of first injections — immune activation response. Expected and transient. Reduce frequency if severe.
moderate VIP hypotension — VIP is a vasodilator. At high doses or in sensitive patients: lightheadedness, blood pressure drop. Titrate slowly. Sit down after administration initially.
moderate VIP immune overstimulation in autoimmunity — In patients with active autoimmune conditions, VIP-mediated immune modulation requires specialist oversight. Not a DIY protocol for autoimmune disease.
low BPC-157 GI effects at high doses — Loose stools or nausea at doses above 1mg twice daily. Reduce dose. Take with food.
🔬 Blood Work · Post-COVID / CIRS Protocol
Before StartingCIRS-specific baseline
CIRS Biomarker Panel Once — specialist lab
  • TGF-β1 — neuroinflammation marker
  • MMP-9 — matrix metalloproteinase
  • C4a — complement activation
  • MSH — melanocyte stimulating hormone
  • VIP (endogenous level) — before supplementing
  • VEGF — vascular endothelial growth factor
  • ADH + osmolality
Standard Safety Panel Once
  • Full blood count
  • Metabolic panel
  • Thyroid
  • Vitamin D
  • Blood pressure (VIP monitoring)
During ProtocolEvery 4–6 weeks
CIRS Response Markers Every 4–6 weeks
  • TGF-β1 — should trend down
  • MMP-9 — tracks neuroinflammation
  • C4a — biotoxin exposure
  • MSH — should rise with VIP
  • VIP endogenous level
Safety Monitoring Monthly
  • Blood pressure — VIP is a vasodilator
  • Symptom severity score (CIRS-validated tool)
  • Visual contrast sensitivity (VCS) test — free online
12-Week AssessmentEnd of initial course
Full CIRS Reassessment Once
  • Complete CIRS biomarker repeat
  • Symptom severity comparison to baseline
  • VCS test repeat
  • Decision: continue, extend, or maintain
♀/♂ Note: CIRS affects men and women equally. No dose differences for Thymosin α-1 or BPC-157. VIP: women may experience more pronounced blood pressure effects — start at 25mcg once daily before titrating up.
Clinic — 02
Metabolic Syndrome Protocol
GLP-1 Agonist · Tesamorelin · 5-Amino-1MQ · Comprehensive Metabolic Reset
The modern functional medicine approach to metabolic syndrome — using GLP-1 receptor agonists as the foundation (with the strongest evidence base in obesity medicine), Tesamorelin for visceral fat reduction, and 5-Amino-1MQ for NAD+ and thermogenic metabolic support. Designed to address insulin resistance, visceral adiposity, and the underlying mitochondrial dysfunction simultaneously.
Physician SupervisedRisk: Moderate
Stack — Compounds & Doses
Semaglutide or Tirzepatide
Sema: 0.25mg → 2.4mg weekly SubQ · Tirz: 2.5mg → 15mg weekly SubQ
Foundation of the protocol. Titrate slowly to minimise GI adverse effects. Semaglutide: start 0.25mg, increase by 0.25mg every 4 weeks. Tirzepatide: start 2.5mg, increase by 2.5mg every 4 weeks. Physician prescription required.
→ Entry page
Tesamorelin
1–2mg SubQ · daily · physician prescribed
Specifically targets visceral fat — the most metabolically dangerous adipose depot. FDA-approved for visceral fat reduction. Synergises with GLP-1 agents: GLP-1 reduces total fat mass; Tesamorelin specifically addresses the visceral component.
→ Entry page
5-Amino-1MQ
50mg oral · daily with breakfast
Supportive — NNMT inhibition raises NAD+, activates thermogenic brown fat programme, improves insulin sensitivity via SIRT1. Addresses the underlying mitochondrial dysfunction common in metabolic syndrome. No prescription required.
→ Entry page
Berberine
500mg oral · 2–3x daily with meals
Essential GLP-1 companion for insulin resistance management. Activates AMPK, reduces hepatic glucose production, improves insulin sensitivity. Mitigates the glucose elevation risk from Tesamorelin.
Protocol & Timeline
Month 1–2GLP-1 titration phase. Prioritise tolerability — GI adverse events at this stage determine compliance. Add 5-Amino-1MQ and berberine from day 1. Tesamorelin added after GLP-1 dose stabilised.
Month 2–4Weight loss accelerates as GLP-1 reaches therapeutic dose. Tesamorelin visceral fat reduction becomes visible — waist circumference the key metric. Monitor fasting glucose monthly given dual glucose-affecting mechanisms.
Month 4–6Body composition stabilisation. DEXA scan recommended at 6 months to objectively measure fat mass, lean mass, and visceral fat changes. Adjust protocol based on findings.
Long-termGLP-1 agents are long-term medications — weight regains on discontinuation. Tesamorelin: continue while visceral fat remains elevated. 5-Amino-1MQ: cycle 12 weeks on, 4 off. Reassess annually.
⚠ Documented Risks & Overdose Concerns
moderate GLP-1 GI adverse events — Nausea, vomiting, diarrhoea — most common cause of discontinuation. Slow titration is the primary mitigation. Antiemetics rarely needed but available. Improves with time.
serious Pancreatitis (class risk) — Rare but documented GLP-1 class risk. Symptoms: severe epigastric pain radiating to back, nausea, elevated lipase. Discontinue immediately if suspected. Risk elevated with prior pancreatitis history — relative contraindication.
moderate Rapid weight loss gallstones — Rapid significant weight loss (>1kg/week) increases gallstone formation risk. Ursodeoxycholic acid 300mg daily may be prescribed prophylactically in high-risk patients. Ensure adequate fat intake to maintain gallbladder emptying.
moderate Tesamorelin glucose elevation — GHRH analogue raises GH → insulin antagonism. Monitor fasting glucose monthly. Berberine co-administration typically manages this without additional intervention.
low Muscle mass preservation — Rapid weight loss risks lean mass loss. Resistance training and protein intake ≥1.6g/kg/day are essential. Some physicians add Ipamorelin/CJC to preserve GH signalling and lean mass during aggressive fat loss.
🔬 Blood Work · Metabolic Syndrome Protocol
Before StartingFull metabolic baseline
Core Metabolic Panel Once
  • HbA1c — primary glycaemic marker
  • Fasting glucose + insulin
  • HOMA-IR — calculated insulin resistance
  • Full lipid panel (LDL, HDL, triglycerides, apoB)
  • Liver function (ALT, AST, GGT) — NAFLD common
Weight & Body Composition Once
  • Weight, BMI, waist circumference
  • DEXA scan (gold standard) if available
  • Blood pressure
Endocrine Panel Once
  • Thyroid (TSH)
  • Fasting leptin
  • Full hormonal panel
During ProtocolMonthly then quarterly
GLP-1 Safety Monthly
  • Fasting glucose — monthly during titration
  • HbA1c — every 3 months
  • Lipase + amylase — if any abdominal pain
  • Blood pressure
Progress Markers Monthly
  • Weight + waist circumference
  • Liver enzymes (ALT, AST) — quarterly
  • Lipid panel — quarterly
At 6 MonthsMajor reassessment
Comprehensive Review Once at 6 months
  • DEXA scan — objective body composition comparison
  • Full metabolic panel repeat
  • Gallbladder ultrasound if rapid weight loss
  • Thyroid repeat
♀ Female note: Women reach therapeutic GLP-1 dose ~2 weeks faster and report higher nausea rates — titrate more slowly. Women with PCOS: include LH, FSH, and testosterone in baseline panel.
Clinic — 03
HGH Clinic Protocol
Physician-Prescribed Somatropin · Monitored Replacement or Anti-Ageing
Prescribed HGH (Somatropin) as used in legitimate clinical practice — from confirmed GH deficiency replacement (FDA-approved) through to the functional medicine anti-ageing context (off-label). Doses are substantially lower than biohacking protocols and require structured monitoring. The safest approach to exogenous HGH.
Physician SupervisedRisk: Moderate — dose and duration dependent
Stack — Compounds & Doses
Somatropin (prescribed)
GHD replacement: 0.2–0.6 IU/day · Anti-ageing: 0.5–1.5 IU/day SubQ
FDA-approved for GH deficiency, HIV wasting, short bowel syndrome. Off-label anti-ageing use requires physician prescription and monitoring. Doses for anti-ageing are significantly lower than both GHD replacement and bodybuilding protocols.
→ Entry page
Secretagogue support (optional)
Sermorelin 300mcg SubQ nightly or Ipamorelin/CJC 200/100mcg 2x/day
Some anti-ageing physicians combine low-dose HGH with secretagogues to preserve pituitary function and maintain some pulsatility alongside exogenous GH. Mechanistically sound — evidence limited.
→ Entry page
Protocol & Timeline
Month 1Start at lowest clinical dose. GHD replacement: per physician calculation based on weight and IGF-1 levels. Anti-ageing: 0.5 IU/day. Baseline IGF-1 blood test essential before starting.
Month 2–3Retest IGF-1. Target: mid-normal for age (typically 150–250 ng/mL for adults over 40, not upper normal). If IGF-1 above upper limit — reduce dose. If sub-therapeutic — cautious increase.
Month 3–6Assess body composition, energy, sleep, and skin quality. DEXA scan at 6 months. Most clinical effects (lean mass, visceral fat reduction, skin quality) require 3–6 months to manifest.
OngoingAnnual monitoring: IGF-1, fasting glucose, HbA1c, blood pressure, haematology. Every 6 months in first year. Dose titration based on IGF-1 levels and clinical response. Regular review with prescribing physician.
⚠ Documented Risks & Overdose Concerns
moderate Carpal tunnel syndrome — Tingling/numbness in hands — first warning sign of excess dose. Reduce by 50%, reassess 4 weeks. Usually resolves. Risk is dose and individual sensitivity dependent.
moderate Insulin resistance — GH is diabetogenic at supraphysiological doses. Even at clinical doses in susceptible individuals. Monitor fasting glucose monthly. Contraindicated in uncontrolled T2D.
serious Acromegaly risk at high doses — Irreversible jaw, hand, and organ enlargement at sustained high doses. Clinical doses with proper IGF-1 monitoring carry very low risk. Never exceed doses that push IGF-1 above upper limit of normal range.
serious IGF-1 and cancer promotion — Supraphysiological IGF-1 promotes cellular proliferation. Annual comprehensive health screening is non-negotiable. Contraindicated with any active malignancy. Relative contraindication with strong family cancer history.
low Oedema / joint pain — Fluid retention and joint ache common in first 4–6 weeks. Dose reduction or dose-on/dose-off (5 days on, 2 off) strategies help. Usually self-resolving.
moderate Prescribing legitimacy — Anti-ageing HGH is off-label in most jurisdictions. Ensure prescribing physician has appropriate expertise and monitoring infrastructure. Grey-market HGH carries adulteration and potency risks.
🔬 Blood Work · HGH Clinic Protocol
Before StartingMandatory pre-treatment
Primary Safety Panel Once — mandatory
  • IGF-1 — treatment target depends on this baseline
  • Fasting glucose + HbA1c
  • Full liver panel
  • Full blood count + haematocrit
  • Kidney function (eGFR)
Cardiovascular Once
  • Lipid panel
  • Blood pressure
  • Resting ECG (if over 40)
Endocrine Once
  • Thyroid (TSH, T3, T4) — HGH affects T4→T3 conversion
  • Cortisol morning level
  • PSA (men over 40)
  • Mammography (women over 40)
Months 1–3Monthly monitoring
IGF-1 Titration Panel Monthly
  • IGF-1 — titrate to maintain mid-normal range for age
  • Fasting glucose
  • Blood pressure
  • Symptom check: carpal tunnel, joint ache, oedema
Ongoing StableEvery 3–6 months
Quarterly Panel Every 3 months
  • IGF-1
  • Fasting glucose + HbA1c
  • Full blood count + haematocrit
Annual Comprehensive Annually
  • Full metabolic, liver, kidney panel
  • Thyroid
  • PSA (men) / Mammography (women)
  • DEXA bone density
  • Comprehensive cancer screening
♀ Female note: Women require 0.2–0.8 IU/day vs 0.5–2 IU/day in men. Female IGF-1 target: 120–230 ng/mL. Monthly bloods for first 6 months. Mammography annually — non-negotiable.
Clinic — 04
TRT Full Clinic Protocol
Testosterone · Gonadorelin · Aromatase Management · Comprehensive Monitoring
The complete physician-supervised testosterone replacement therapy protocol — from testosterone delivery through HPG axis preservation, oestrogen management, and comprehensive blood monitoring. Designed for long-term sustainable hormone optimisation rather than maximum anabolism.
Physician SupervisedRisk: Low when monitored
Stack — Compounds & Doses
Testosterone Cypionate or Enanthate
80–200mg IM or SubQ · weekly (preferred) or fortnightly
Weekly SubQ injections preferred over fortnightly IM — more stable testosterone and oestradiol levels, fewer peaks and troughs, easier self-administration. Start at conservative dose (80–100mg/week) and titrate based on blood levels to target mid-normal range (500–800 ng/dL total testosterone).
Gonadorelin
100–200mcg SubQ · twice daily
Maintains HPG axis pituitary connection — LH and FSH remain measurable, testicular function preserved. ~$15–20/month from compounding pharmacy. The key TRT companion since HCG reclassification.
→ Entry page
Anastrozole (if needed)
0.25–0.5mg · twice weekly · only if E2 confirmed elevated
Not everyone needs an AI — many physicians now take a minimisation approach, adding only if oestradiol confirmed elevated on blood test AND patient is symptomatic. Over-suppression of oestrogen causes its own problems (bone density, mood, lipids).
HCG (fertility-focused patients)
500–1500 IU SubQ · 2–3x weekly · if gonadorelin inadequate
For patients prioritising fertility preservation where gonadorelin response is inadequate. HCG directly stimulates Leydig cells — more reliably maintains intratesticular testosterone and spermatogenesis. Requires prescription.
Protocol & Timeline

Blood test targets: Total testosterone 500–800 ng/dL (mid-normal); oestradiol 20–35 pg/mL; haematocrit below 52%; PSA within age-appropriate range; LH detectable (confirms gonadorelin working).

Dose timing: Test blood 48 hours after last injection (trough) for most accurate assessment of lowest sustained level. Avoid testing at peak (24–36h post-injection) — will overestimate average level.

Long-term considerations: Haematocrit elevation (polycythaemia) is the most clinically significant long-term risk — increases blood viscosity and clot risk. Therapeutic phlebotomy (blood donation) effectively manages this. Some physicians use lower-dose more-frequent injections (daily SubQ microdosing) to minimise haematocrit elevation.

⚠ Documented Risks & Overdose Concerns
moderate Haematocrit elevation / polycythaemia — Most clinically significant long-term TRT risk. Haematocrit above 52% increases blood viscosity and venous thromboembolism risk. Monitor every 3 months. If elevated: reduce testosterone dose, increase injection frequency, or consider therapeutic phlebotomy (blood donation).
moderate Oestrogen imbalance — Both excess and deficient oestrogen cause problems. Excess: gynaecomastia, water retention, mood instability. Deficient (over-AI use): bone density loss, joint pain, poor lipid profile, low libido. Monitor E2 and treat to symptom resolution, not arbitrary numbers.
low Testicular atrophy — Without gonadorelin or HCG, testicular volume decreases over months-years on TRT. Gonadorelin twice daily mitigates but does not completely prevent this in all patients. Cosmetically significant for some patients.
low PSA elevation — Testosterone does not cause prostate cancer but may accelerate pre-existing cancer. Baseline PSA essential before starting TRT. Annual PSA monitoring in patients over 45. Rapid PSA rise is a red flag.
low Infertility — TRT suppresses spermatogenesis. Gonadorelin partially preserves FSH and spermatogenesis but does not reliably maintain fertility in all patients. For fertility-focused patients: consider HCG + FSH supplementation or sperm banking before starting TRT.
🔬 Blood Work · Male TRT Full Protocol
Before StartingMandatory — trough timing (48h post-injection)
Full Testosterone Panel Once
  • Total testosterone
  • Free testosterone
  • LH + FSH — suppression level
  • Oestradiol E2 — sensitive assay
  • SHBG
  • Prolactin
  • PSA (men over 45)
Safety Panel Once
  • Full blood count + haematocrit
  • Metabolic panel
  • Lipid panel
  • Blood pressure
During ProtocolQuarterly — always trough
Core TRT Panel Every 3 months
  • Total testosterone — target 500–800 ng/dL
  • Oestradiol E2 — target 20–35 pg/mL
  • Haematocrit — must stay below 52%
  • LH + FSH — confirms gonadorelin working
  • PSA (men over 45)
Annual Additions Annually
  • Free testosterone
  • SHBG
  • Full metabolic panel
  • Lipid panel
  • DEXA bone density (if on TRT >2 years)
Fertility GoalEvery 6 months if applicable
Fertility Panel Every 6 months
  • Semen analysis — count, motility, morphology
  • FSH — spermatogenesis activity
  • Inhibin B — Sertoli cell function
  • LH — pulsatile GnRH response
♂ Male TRT — see Clinic 04b for Female Testosterone Replacement.
Clinic — 04b
Female Testosterone Replacement
Low-Dose Female TRT · Compounded Testosterone · Perimenopause & Menopause
Women lose testosterone from the mid-30s — often before oestrogen decline is noticeable. Female TRT addresses libido, energy, lean mass, cognitive clarity, mood stability, and bone density at doses 10–20× lower than male TRT. Profoundly effective when dosed correctly. Unique androgenic risks require slower titration and sex-specific monitoring.
Physician Supervised ♀ Female Specific Risk: Low when monitored
♂ Male TRT Reference Dose
80–200mg/week Testosterone Cypionate
Target total T: 500–800 ng/dL · Oestradiol: 20–35 pg/mL · Haematocrit primary concern · Test 48h post-injection (trough)
♀ Female TRT Dose
5–20mg/week Testosterone Cypionate SubQ
Target free testosterone mid-normal for age (1–3 ng/dL free T) · Total T: 30–80 ng/dL upper-normal female range · SHBG critical — high SHBG binds T and reduces free fraction · Test at same cycle day each time (premenopausal)
Stack — Compounds & Female Doses
Testosterone Cypionate (injectable)
♀ 5–20mg SubQ weekly · start 5mg · titrate 2.5mg every 4–6 weeks
Preferred for precise dosing and stable levels. Weekly SubQ into abdomen or thigh. Most women find their optimal range at 8–15mg/week. Takes 6–12 weeks to reach stable blood levels. Faster libido response than men — often noticeable within 2–4 weeks vs 6–8 weeks.
Compounded Testosterone Cream (alternative)
♀ 1–5mg daily topical · inner wrist or inner labia
Needle-free option. Absorption varies significantly between individuals — harder to titrate precisely than injectable. Inner labia gives higher bioavailability than wrist. Avoid skin-to-skin contact with partners until dry. Do not apply to areas where children may touch.
Kisspeptin-10 (libido support)
♀ 50mcg SubQ · 2x weekly · optional addition
Direct central libido enhancement via GPR54 limbic receptors — independent of testosterone levels. Valuable when libido improvement from testosterone alone is insufficient. Also supports GnRH signalling in perimenopausal women with declining hypothalamic drive.
→ Entry page
DHEA (if DHEA-S low on bloodwork)
♀ 5–25mg oral · daily morning · physician prescribed
Precursor to both testosterone and oestrogen — useful in women with low DHEA-S (common post-menopause). Acts synergistically with TRT. Topical DHEA (Intrarosa) FDA-approved for vaginal atrophy. Test DHEA-S at baseline — supplement only if confirmed low.
Protocol, Timeline & Female-Specific Considerations
BaselineEssential before starting: free testosterone (critical — not just total), total testosterone, SHBG, oestradiol, FSH/LH, DHEA-S, full blood count, lipids, HbA1c, thyroid. Premenopausal women: test on days 8–12 of menstrual cycle for consistent baseline — testosterone varies across the cycle.
Week 1–6Start at 5mg/week injectable or 1mg/day cream. Libido improvement often first and most rapid effect in women — 2–4 weeks. Also: energy, mood, sleep quality. Monitor for androgenic signs: acne, hair changes at temples, clitoral sensitivity changes, any voice changes.
Week 6–8First blood recheck: free testosterone (most important), total T, SHBG. If free T below mid-normal and symptoms persist — increase by 2.5–5mg/week. If any androgenic signs — reduce dose. Premenopausal: retest at same cycle day.
Month 3–6Full reassessment. Most women stable at target dose by month 3. Bone density, lean mass, cognitive effects take longer — assess formally at 6 months. DEXA recommended if post-menopausal or if bone density was a starting concern.
OngoingQuarterly bloods first year, then every 6 months when stable. Annual comprehensive panel, mammography per age guidelines. Female TRT is typically a long-term therapy — most women continue indefinitely once optimised.
♀ Key Differences from Male TRT
Libido responds faster — 2–4 weeks vs 6–8 in men. SHBG is critical — high SHBG (common in women, especially those on oral contraceptives or oral oestrogen HRT) binds testosterone and lowers the free fraction. If total T looks normal but symptoms persist — check free T and SHBG. Test timing matters in premenopausal women — always test at the same menstrual cycle phase. Oestrogen interplay — testosterone works best alongside adequate oestrogen and progesterone in peri/post-menopause; testosterone without oestrogen often gives suboptimal results. Voice changes are irreversible — the only androgenic side effect that cannot be reversed. Stop immediately if any voice changes occur.
⚠ Female-Specific Risks — With Mitigations
serious Voice deepening — irreversible — The one androgenic side effect that cannot be reversed after it occurs. Vocal cord thickening is permanent. Mitigation: start at absolute minimum dose (5mg/week), titrate slowly, stop immediately at first sign of any voice change. This is why slow titration is non-negotiable in female TRT.
moderate Clitoral enlargement — Dose-dependent, may become permanent at excess doses. Minor changes at therapeutic doses are common and typically well-tolerated. Mitigation: keep free testosterone within mid-normal female range. Do not chase "more is better." Reduce dose if changes exceed comfort.
moderate Acne — DHT-mediated. Common at higher doses or in DHT-sensitive individuals. Mitigation: zinc 30mg/day reduces DHT conversion. Topical retinoids. Ensure total testosterone does not exceed upper-normal female range. Check DHT levels if persistent.
moderate Androgenic hair loss — DHT-sensitive women may experience temporal hair thinning. Mitigation: check DHT if occurs. Low-dose finasteride 0.5mg/day reduces DHT conversion. Topical minoxidil. Reduce testosterone dose. Ketoconazole shampoo reduces scalp DHT locally.
low Haematocrit elevation — Less common than male TRT at these doses but still monitor. Mitigation: regular haematocrit monitoring, stay hydrated, reduce dose if elevated above 48% in women.
low Menstrual cycle changes — Testosterone may affect cycle regularity in premenopausal women. Discuss fertility implications before starting. Mitigation: use lowest effective dose. Discuss family planning before starting. Sperm banking or embryo freezing if fertility is a priority.
🔬 Blood Work · Female TRT — Full Schedule
Before Starting Premenopausal: test days 8–12 of cycle
Full Testosterone Panel Once
  • Free testosterone — primary marker (not total)
  • Total testosterone
  • SHBG — critical: high SHBG reduces free T
  • Oestradiol E2
  • FSH + LH — menopausal status
Supporting Panel Once
  • DHEA-S
  • Full blood count + haematocrit
  • Lipid panel
  • HbA1c + fasting glucose
  • Liver function
  • Thyroid (TSH + free T3/T4)
  • DHT (if acne/hair concerns at baseline)
Screening Once
  • Mammography (over 40)
  • Cervical smear (if due)
  • Blood pressure
Weeks 6–8 First titration check
Dose Titration Panel Week 6–8
  • Free testosterone — most important
  • Total testosterone
  • SHBG
  • Oestradiol — HRT interplay
  • Haematocrit
Month 3 & 6 Stabilisation monitoring
Month 3 Panel Month 3
  • Full testosterone panel (free T, total T, SHBG)
  • DHT — add if acne or hair thinning developing
  • Lipids
  • Full blood count
Month 6 Panel Month 6
  • Complete repeat of baseline panel
  • DEXA bone density (if post-menopausal or on HRT)
  • Mammography if due
Ongoing Stable Every 6 months
Maintenance Panel Every 6 months
  • Free testosterone + SHBG
  • Total testosterone
  • Haematocrit
  • Oestradiol
  • Lipids
Annual Panel Annually
  • Full comprehensive blood panel
  • Thyroid
  • DHT (if any androgenic symptoms)
  • Mammography
  • Cervical smear
  • DEXA (if post-menopausal)
  • Cardiovascular risk assessment
♀ Key reminder: Always test at the same point in your menstrual cycle (premenopausal). The number that matters most is free testosterone — not total. High SHBG can make total testosterone look normal while free testosterone is very low. Test SHBG whenever you test testosterone.
Clinic — 05
Anti-Ageing Comprehensive Panel
Sermorelin · Thymosin α-1 · GHK-Cu · Epitalon · Collagen
The functional medicine anti-ageing protocol — addressing GH axis decline (Sermorelin), immune senescence (Thymosin α-1), tissue quality (GHK-Cu and Collagen), and telomere/longevity signalling (Epitalon). Designed for patients over 40 wanting to address the mechanisms of biological ageing comprehensively.
Physician SupervisedRisk: Low
Stack — Compounds & Doses
Sermorelin
300mcg SubQ · nightly · long-term
GHRH analogue — stimulates the pituitary to produce GH naturally. Preserves pulsatility. Gentler and more physiological than exogenous HGH. FDA-approved for paediatric GH deficiency; widely used off-label for adult anti-ageing. Long-term use well tolerated.
→ Entry page
Thymosin α-1
1.5mg SubQ · twice weekly · 12-week courses
Addresses immune senescence — the decline in T-cell function and NK cell activity central to biological ageing and increased infection/cancer susceptibility. Run 12-week courses 2x/year or continuous at physician direction.
→ Entry page
GHK-Cu
Topical 1% daily + 0.5–1mg SubQ · 3x/week
Drives collagen synthesis, activates over 4,000 genes, powerful anti-inflammatory and anti-oxidant effects. Topical for facial skin; SubQ for systemic gene regulation. One of the best-evidenced cosmetic and systemic anti-ageing peptides.
→ Entry page
Epitalon
5–10mg SubQ or IV · 10-day course · twice yearly
Pineal gland tetrapeptide — activates telomerase, regulates circadian rhythm, documented life extension in animal models. Intensive twice-yearly courses rather than continuous dosing. Some physicians administer IV for the telomerase activation course.
→ Entry page
Collagen Peptides (oral)
10–15g daily · hydrolysed · Type I/II/III
The accessible cornerstone — strong RCT evidence for skin elasticity, joint pain reduction, and bone density support. Not glamorous but among the best-evidenced compounds in anti-ageing medicine. Add Vitamin C 500mg to maximise collagen synthesis.
→ Entry page
Protocol & Timeline

Morning: Collagen peptides + Vitamin C. GHK-Cu topical.

Evening: Sermorelin SubQ (nightly). GHK-Cu SubQ 3x/week.

Twice weekly: Thymosin α-1 1.5mg SubQ.

Twice yearly (10-day courses): Epitalon 5–10mg/day SubQ or IV.

Patient education: Anti-ageing protocols require 3–6 months minimum for meaningful assessment. Set appropriate expectations — these are not acute-effect compounds. Annual biological age testing (epigenetic clock, telomere length) is the most objective outcome measure.

⚠ Documented Risks & Overdose Concerns
low Sermorelin water retention and joint ache — Common in first 2–4 weeks at initiation. Self-resolving. Dose reduction if persistent. Monitor IGF-1 at 8 weeks — ensure not supraphysiological.
low Thymosin α-1 immune activation response — Fatigue, mild flu-like symptoms after first 1–2 injections. Immune system activation. Expected, transient. Reduce frequency to once weekly initially if severe.
low Epitalon telomerase concern — Theoretical: telomerase activation in cancer cells. Epitalon has anti-tumour activity in animal models and is considered safe. Contraindicated with active malignancy as standard precaution.
low Autoimmune caution with immune modulators — Thymosin α-1 and GHK-Cu both modulate immune function. In patients with autoimmune conditions, specialist review before starting. Not an absolute contraindication but requires individualisation.
🔬 Blood Work · Anti-Ageing Panel
Before StartingBiological age baseline
Longevity Biomarkers Once
  • IGF-1 — Sermorelin titration target
  • Full blood count + haematocrit
  • Comprehensive metabolic panel
  • Inflammatory panel: hsCRP, IL-6
  • Lipid panel (apoB, Lp(a))
Biological Age Tests Baseline then annually
  • Epigenetic clock (TruAge or equivalent) — primary outcome
  • Telomere length
  • DHEA-S
  • Thyroid full panel
  • Vitamin D
Immune Function Baseline then annually
  • NK cell count + activity
  • CD4/CD8 T-cell ratio
  • Lymphocyte subsets
During ProtocolQuarterly
Core Panel Every 3 months
  • IGF-1 — Sermorelin titration (mid-normal for age)
  • hsCRP — should trend down
  • Fasting glucose
  • Full blood count
Annual ReassessmentOnce per year
Biological Age Comparison Annually
  • Repeat epigenetic clock
  • Telomere length comparison
  • Immune function panel repeat
  • DEXA bone density
  • Comprehensive cancer screening
♀/♂ Note: Sermorelin: women may see stronger IGF-1 response — monitor at 6 weeks and reduce to 200mcg if above target. GHK-Cu: women often see enhanced skin response. Compare epigenetic clock to sex-matched references.
Clinic — 06
Injury Recovery — Compounded
Arg-BPC-157 · TB-500 · Collagen · Vitamin C · Physician Protocol
The clinic version of injury recovery — using legally compounded Arg-BPC-157 (oral, Category 1 from 2025) and TB-500 prescribed through a physician with appropriate monitoring. More conservative doses than biohacking protocols, emphasis on the oral BPC-157 route for patient compliance, and structured with labs and follow-up.
Physician SupervisedRisk: Low
Stack — Compounds & Doses
Arg-BPC-157 (compounded oral)
500mcg twice daily oral · 30 min before meals
FDA Category 1 from 2025 — compounding pharmacies can legally prepare. Oral form preferred in clinic setting for compliance. Higher dose than injectable equivalent to compensate for oral bioavailability difference.
→ Entry page
TB-500 (compounded SubQ)
2mg SubQ · twice weekly loading (4 weeks) → weekly maintenance
Prescribed and compounded. Loading phase 4 weeks, then maintenance. Physician administers first injection and trains patient on self-injection technique.
→ Entry page
Collagen Peptides (oral)
15g daily · Type I + III for tendon/ligament · Type II for cartilage
Clinical-grade hydrolysed collagen as the nutritional foundation. Strong RCT evidence for tendon and joint repair when combined with Vitamin C (essential co-factor for collagen synthesis).
→ Entry page
Vitamin C
500–1000mg oral · with collagen · twice daily
Essential co-factor for collagen hydroxylation. The RCT evidence for collagen + Vitamin C timing is specific — take together 30–60 minutes before exercise or at stable times if not exercising.
Protocol & Timeline
Week 1–4Loading phase. BPC-157 oral twice daily + TB-500 loading 2mg twice weekly. Collagen + Vitamin C daily. Most patients report pain reduction of 30–50% and improved mobility by week 4.
Week 4–8Transition to maintenance. TB-500 once weekly. Continue BPC-157 oral twice daily. Target: full pain-free range of motion. Begin progressive loading physiotherapy alongside peptide protocol.
Week 8–12Assessment at 12 weeks. If target achieved — taper BPC-157 to once daily for 4 more weeks, then discontinue. TB-500 can be discontinued at 12 weeks. Continue collagen indefinitely for structural maintenance.
OngoingCollagen + Vitamin C indefinitely. BPC-157 occasional cycles (4–6 weeks) if re-injury or symptom recurrence. Annual check with prescribing physician.
⚠ Documented Risks & Overdose Concerns
low Oral BPC-157 GI effects — Occasional nausea at initiation. Take with food if sensitive. Resolves within first week.
low TB-500 injection technique — SubQ injection training essential. Improper technique causes unnecessary bruising. First injection always at clinic.
low Combined protocol vs purity concern — Risk is primarily sourcing — compounded peptides from legitimate pharmacies are clean; research chemicals are not. This is the key advantage of the clinic pathway over self-directed biohacking.
🔬 Blood Work · Injury Recovery (Compounded)
Before StartingLight baseline
Basic Panel Once
  • Full blood count
  • hsCRP — baseline inflammation
  • Kidney function — BPC-157 is cleared renally
  • Liver function
During ProtocolWeeks 4 and 12
Progress Monitoring Weeks 4 and 12
  • hsCRP — trending down confirms anti-inflammatory action
  • Pain score (VAS 0–10) — weekly self-reported
  • Range of motion — physiotherapist measured
  • Imaging (ultrasound/MRI) at 12 weeks if available
Post-Protocol4–6 weeks after
Final Assessment Once
  • hsCRP — confirm return to baseline
  • Functional movement screen
  • Return to sport/activity assessment
♀/♂ Note: No significant dose differences for Arg-BPC-157 or TB-500. Women and men heal at similar rates. Female athletes: healing can slow during luteal phase — expected variation, not a protocol concern.
Clinic — 07
Female Hormonal & Menopause Protocol
Kisspeptin-10 · Bremelanotide · Oxytocin · Low-Dose HGH
A specialist physician-supervised protocol addressing the multiple dimensions of menopausal and perimenopausal decline — the GnRH/LH axis (Kisspeptin), sexual desire (Bremelanotide PT-141), bonding and mood (Oxytocin), and tissue/metabolic quality (low-dose HGH). Women require lower HGH doses than men and are more sensitive to both benefits and side effects.
Physician SupervisedRisk: Moderate — specialist required
Stack — Compounds & Doses
Kisspeptin-10
50–100mcg SubQ · 2x weekly
Addresses the upstream HPG axis changes in perimenopause. Kisspeptin neurons decline with age, contributing to reduced GnRH pulsatility. Also has direct effects on sexual motivation via limbic GPR54 receptors independent of oestrogen. Growing evidence base in female sexual dysfunction.
→ Entry page
Bremelanotide PT-141
1.25–1.75mg SubQ · 45 min before desired activity · as needed
FDA-approved for hypoactive sexual desire disorder (HSDD) in premenopausal women. MC4R agonist — central sexual motivation mechanism. Start at 1.25mg, assess tolerance for nausea and blood pressure effects before using 1.75mg.
→ Entry page
Oxytocin (intranasal)
16–24 IU intranasal · as needed or before intimate situations
The bonding and trust neuropeptide — addresses the emotional intimacy and social connection dimensions of sexual health that Bremelanotide does not. Used 20–30 min before desired effect. Some physicians prescribe for chronic anxiety and emotional dysregulation in menopause.
→ Entry page
Low-Dose HGH (if indicated)
0.2–0.5 IU/day SubQ · physician prescribed · monitored
The lowest effective range for body composition and tissue quality in women. Women are significantly more sensitive to HGH than men — start at 0.2 IU and titrate based on IGF-1 levels. Effects: visceral fat reduction, lean mass, skin quality, energy, sleep. Must not be used without monitoring.
→ Entry page
Protocol & Timeline

Context with HRT: This protocol is typically used alongside or after conventional hormone replacement therapy (oestrogen ± progesterone), not instead of it. Peptides address the dimensions of hormonal health that conventional HRT does not fully cover.

Kisspeptin + sexual function: Kisspeptin-10 has the unique property of enhancing sexual motivation through both peripheral hormonal mechanisms (GnRH→LH→oestrogen) and direct limbic circuit activation. In multiple human studies it improved sexual function scores in women with hypoactive sexual desire disorder.

Bremelanotide practical note: Nausea is the most common side effect (40% at 1.75mg). Take anti-nausea medication (ondansetron or domperidone) 30 min before if sensitive. Blood pressure rises transiently — avoid in uncontrolled hypertension.

HGH in women: Doses above 0.5 IU/day are rarely needed and carry disproportionate side effect risk in women. The target IGF-1 level in women is the same mid-normal range as men but requires lower doses to achieve it. Monthly monitoring is essential in the first 6 months.

⚠ Documented Risks & Overdose Concerns
moderate Bremelanotide nausea and blood pressure — Nausea in ~40% at 1.75mg — pre-medicate with anti-nausea medication if sensitive. Transient BP elevation — absolute contraindication in uncontrolled hypertension or cardiovascular disease.
moderate HGH in women — higher sensitivity — Women experience side effects (carpal tunnel, fluid retention, glucose effects) at lower doses than men. Start at 0.2 IU, titrate cautiously. Monthly IGF-1 monitoring mandatory.
low Oxytocin and mood effects — Occasional paradoxical anxiety or emotional intensity at high doses. Reduce to 8–16 IU if this occurs. Generally very well tolerated.
low Kisspeptin oestrogen stimulation — Kisspeptin increases LH and downstream oestrogen in premenopausal women. In postmenopausal women or those on oestrogen-sensitive cancer therapy, specialist review required before use.
🔬 Blood Work · Female Hormonal & Menopause
Before StartingComprehensive female baseline
Reproductive Hormones Once — premenopausal: days 8–12 of cycle
  • LH + FSH — menopausal status
  • Oestradiol E2
  • Progesterone (days 19–22 if premenopausal)
  • Total + free testosterone
  • SHBG
  • DHEA-S
  • Prolactin
  • AMH — ovarian reserve
Safety & Metabolic Once
  • Full blood count + metabolic panel
  • Thyroid full panel
  • Lipids + blood pressure
  • Mammography (over 40)
  • Bone density DEXA (if post-menopausal)
During ProtocolRegular monitoring
Hormonal Tracking Every 6–8 weeks
  • Oestradiol — HGH + Kisspeptin can affect oestrogen
  • Free testosterone (if on female TRT alongside)
  • Blood pressure — PT-141 + Kisspeptin monitoring
  • SHBG — affects free hormone levels
HGH Monitoring (if included) Monthly
  • IGF-1 — target 120–230 ng/mL for women
  • Fasting glucose — monthly at any HGH dose
  • Carpal tunnel symptoms — self-reported
Ongoing StableEvery 6 months
Comprehensive Female Panel Every 6 months
  • Full reproductive hormone panel
  • IGF-1 (if on HGH)
  • Lipids + metabolic panel
  • Mammography (annual)
  • DEXA bone density (annual if post-menopausal)
  • Cervical smear per guidelines
Clinic — 08 · Cross-Community
Tissue Repair — Clinic Protocol
Compounded BPC-157 · TB-500 · The Wolverine Stack in Clinical Practice
The biohacking community's most popular stack — validated and adopted by functional medicine clinics. The compounds, mechanism, and outcomes are the same; the clinic version uses legally compounded preparations, physician oversight, conservative doses, and structured monitoring rather than self-directed research chemical use. One of the clearest examples of community-to-clinic knowledge transfer in peptide medicine.
Physician Supervised Cross-Community ↔ Risk: Low
Stack — Compounds & Doses
Arg-BPC-157 (oral compounded)
500mcg–1mg oral · twice daily · 30 min before meals
FDA Category 1 from 2025 — compounding pharmacies can legally dispense. Oral route preferred in clinic setting — better patient compliance than injection for a long-course protocol. Higher dose compensates for oral bioavailability vs injectable.
→ Entry page
TB-500 (compounded SubQ)
2mg SubQ · twice weekly × 4 weeks loading → weekly maintenance
Physician-compounded and dispensed. First injection training at clinic. TB-500 distributes systemically — less site-specific than BPC-157. The combination covers both local (BPC-157) and systemic (TB-500) tissue repair mechanisms simultaneously.
→ Entry page
Collagen Peptides + Vitamin C
15g hydrolysed collagen + 500mg Vitamin C · daily
Nutritional foundation — RCT evidence for tendon and ligament repair when combined with Vitamin C. Often the only component patients continue indefinitely after the peptide course completes. Type I + III for tendon/muscle; Type II for cartilage.
→ Entry page
Community vs Clinic — Key Differences

What's the same: The core compounds (BPC-157 + TB-500), the mechanisms (VEGFR2 angiogenesis + actin regulation), and the outcomes (pain reduction, mobility restoration, accelerated healing) are identical. Both communities converge on 8–12 week courses as the sweet spot. Both use Collagen + Vitamin C as structural support.

What's different: The clinic uses oral Arg-BPC-157 (more compliant, legally compounded) rather than injectable BPC-157 acetate. Doses are slightly more conservative. There's structured follow-up with physiotherapy integration. Lab safety checks at baseline and 6 weeks.

Why clinics adopted it: The biohacking community generated years of consistent outcome reports before functional medicine physicians started incorporating this stack. The community essentially ran an uncontrolled longitudinal study that clinicians observed and eventually incorporated into practice. The 2025 FDA Category 1 reclassification of BPC-157 legitimised the clinical pathway.

Timeline: Both communities report similar timelines — 30–50% pain reduction by weeks 4–6, functional restoration by weeks 8–12. The consistency of this timeline across thousands of independent reports (biohacker and clinic) is one of the strongest cross-community consensus signals for any protocol in this book.

⚠ Documented Risks & Overdose Concerns
low Oral BPC-157 GI tolerance — occasional nausea at initiation, resolves in first week. Take with food if sensitive. No serious adverse events documented at standard doses in either community.
low TB-500 injection site reactions — minor redness and swelling. Self-resolving. First injection at clinic with training reduces patient anxiety and technique errors.
low Wide therapeutic window — BPC-157 has no established toxicity ceiling in animal models. No documented acute toxicity from dose escalation in human use across either community. The stack's safety profile is one reason for its broad adoption.
moderate Theoretical proliferative concern (long-term) — BPC-157 promotes growth factor expression. Not observed clinically at standard course lengths. Contraindicated with active or recent malignancy as standard precaution.
📋 Monitor / Track
Pain scores (VAS — weekly) Range of motion (physio measured) Return to function date Injection site assessment Physiotherapy integration milestones
Section Three

Cross-Community Consensus

Where biohackers and clinic patients independently report the same outcomes — the strongest signal in this book

These five findings have emerged independently from both the biohacking community (self-directed, research compounds) and peptide clinic patients (physician-supervised, monitored). When two populations using different doses, different sourcing, different monitoring, and different starting conditions consistently report the same outcomes — that convergence is the most meaningful signal in peptide science that does not yet have large-scale RCT validation.

BPC-157 + TB-500 Tissue Repair Stack
Injury recovery · tendon · ligament · muscle · post-surgical healing
Consensus Strength
★★★★★ Very High
Biohacking Community Reports
The Wolverine Stack is the most consistently reported peptide protocol in biohacking history. Years of forum reports, personal blogs, and community discussion converge on the same outcomes: 30–50% pain reduction by weeks 4–6, functional restoration by 8–12 weeks for most injuries, and a qualitative "something is clearly working" experience that distinguishes it from placebo in self-report. It is the compound most frequently described as life-changing by injured athletes and weekend warriors alike. Community protocol: BPC-157 250–500mcg daily SubQ + TB-500 2–2.5mg twice weekly loading then weekly.
Clinic Patient Reports
Functional medicine and regenerative medicine clinics that have adopted this protocol (accelerated by BPC-157's 2025 FDA Category 1 reclassification) report outcomes that match community timelines closely: most patients report meaningful pain reduction within 4 weeks, functional improvement by week 8. Clinic version typically uses oral Arg-BPC-157 (500mcg–1mg twice daily) for compliance ease. Physicians describe it as "the most effective tissue healing protocol we have access to outside PRP and stem cells" — and at a fraction of the cost. Growing integration with physiotherapy programmes.
Consensus Finding
The convergence here is particularly compelling because the two communities use different forms (injectable vs oral), different doses, different injury types, and have no coordinated reporting — yet independently arrive at the same 4–6 week response timeline and 8–12 week restoration timeline. The community essentially ran a years-long uncontrolled observational study that functional medicine physicians then observed and adopted. It represents the clearest example of community-to-clinic knowledge transfer in peptide medicine, and the cross-community agreement on both timeline and outcome is one of the strongest non-RCT signals in this reference.
BPC-157 Gut Healing
Leaky gut · IBS · IBD · Post-antibiotic gut restoration
Consensus Strength
★★★★★ Very High
Biohacking Community Reports
Biohackers with gut issues — leaky gut, IBS, post-antibiotic dysbiosis — consistently report BPC-157 as one of the most impactful compounds they have used. Oral dosing (500mcg–1mg) used for gut-specific applications. Typically report 4–8 weeks to meaningful improvement. The most consistent positive report in the biohacking community across any application.
Clinic Patient Reports
Functional medicine and integrative GI clinics prescribing Arg-BPC-157 (oral, compounded) for leaky gut syndrome and IBD report consistent patient improvement within 4–8 weeks. Physicians describe it as "the most effective gut healing peptide we have access to." Post-COVID gut dysfunction a growing indication.
Consensus Finding
Gut healing is the one BPC-157 application where both communities are in near-complete agreement. The mechanism (VEGFR2-driven angiogenesis, growth factor upregulation, direct mucosal repair) is well-characterised and specific to the GI tract. Oral delivery concentrates the compound at the target tissue before systemic distribution. Both communities prefer oral over injectable for this specific indication.
MK-677 Sleep Quality Improvement
Stage 4 slow-wave sleep · REM enhancement · recovery
Consensus Strength
★★★★★ Very High — RCT confirmed
Biohacking Community Reports
MK-677 sleep improvement is reported so consistently in the biohacking community that it has become the primary reason many users continue taking it beyond body composition goals. Typically noticeable within the first 3–7 days. Described as "the deepest sleep I have had in years." Often maintained even when body composition effects plateau.
Clinic Patient Reports
Functional medicine physicians prescribing MK-677 for anti-ageing and body composition consistently report that sleep improvement is the most appreciated outcome by their patients — often exceeding the body composition changes in patient satisfaction. Many physicians describe starting with sleep improvement as the patient motivation for compliance.
Consensus Finding
The RCT evidence supports this finding — Copinschi 1997 confirmed 50% increase in stage 4 slow-wave sleep and 20%+ REM increase in young adults. The cross-community convergence on this effect is perhaps the strongest validated finding in the book: robust RCT data + consistent biohacker reports + consistent clinic reports all pointing at the same effect.
Thymosin α-1 Immune Response
Chronic infection · post-viral immune dysregulation · immune senescence
Consensus Strength
★★★★☆ High — clinical evidence + community
Biohacking Community Reports
Biohackers using Thymosin α-1 for chronic fatigue, recurring infections, or post-viral immune dysfunction consistently report improved infection resilience and energy. Particularly strong reports from the Long-COVID and CIRS biohacking communities. Many report fewer infections annually and faster recovery when ill.
Clinic Patient Reports
The most consistent peptide outcome in CIRS and post-viral clinics. Physicians using Shoemaker-informed protocols report Thymosin α-1 as the most reliably effective immune reset peptide they prescribe. Post-COVID clinics report consistent improvement in T-regulatory function markers alongside patient-reported symptom reduction.
Consensus Finding
The 35+ country approvals and 30+ clinical trials for Thymosin α-1 give it the strongest formal evidence base of any peptide in the immune space. The convergence of clinic data (monitored, lab-confirmed immune marker improvement) with biohacker reports (consistent subjective immune function improvement) across two completely different populations is a compelling signal.
GHK-Cu Skin and Wound Quality
Skin elasticity · collagen density · wound healing · hair follicle
Consensus Strength
★★★★☆ High — RCT + industry + community
Biohacking Community Reports
GHK-Cu is one of the few peptides where the biohacking community uses both topical and SubQ routes and consistently rates both as effective — topical for facial ageing, SubQ for systemic effects. Skin quality improvement (elasticity, fine lines, texture) reported within 6–12 weeks of consistent use. Hair density improvement reported by a subset of users.
Clinic Patient Reports
Cosmetic dermatologists and functional medicine physicians prescribing GHK-Cu consistently report it as the most evidence-backed topical anti-ageing peptide available. Patient photography at 3 and 6 months routinely shows measurable improvement in skin quality. Used in high-end clinic skincare protocols alongside conventional treatments.
Consensus Finding
GHK-Cu has one of the strongest evidence bases of any cosmetic peptide — used in multiple Strivectin and professional skincare formulations, with RCT evidence for collagen synthesis stimulation and wound healing. The community-clinic convergence here is reinforced by commercial skincare industry validation, making it the most broadly corroborated finding in this section.
Semaglutide / GLP-1 Weight Loss
Sustained appetite suppression · 15%+ weight loss · metabolic reset
Consensus Strength
★★★★★ Very High — Phase 3 RCT + prescribing data + community
Biohacking Community Reports
The biohacking community moved onto GLP-1 agonists (particularly compounded semaglutide during the shortage period) en masse from 2022–2024. Reports are overwhelmingly consistent: sustained appetite suppression with no willpower required, 10–20% weight loss over 6–12 months, with the "food noise" cessation being the most consistently described subjective experience. Considered the most effective compound the community has encountered for weight management.
Clinic Patient Reports
GLP-1 agonists are the fastest-growing prescription category in the history of obesity medicine. Physicians prescribing semaglutide and tirzepatide report outcomes that align precisely with biohacker reports — 15% average weight loss in clinical practice, sustained appetite reduction, and a quality of patient experience that is qualitatively different from previous weight loss medications.
Consensus Finding
The GLP-1 consensus is unique in this section because it also has the largest controlled trial evidence base (STEP 1, SURMOUNT-1) confirming what both communities independently report. This is not a peptide where community reports preceded science — the science and the community reports arrived simultaneously and agree completely. The strongest cross-community-RCT-clinical-practice convergence in the entire book.
Section Five

Highly Experimental Protocols

Frontier compounds · animal data only or minimal human data · serious biohackers only

⚡ Important — Read Before Proceeding

The protocols in this section are documented for educational purposes only. They represent what serious longevity researchers, biohackers, and self-experimenters are actually running — not what is clinically established or recommended. Most of these compounds have no human clinical trials. Evidence comes from animal models, mechanistic data, and community n=1 reports. These are frontier protocols — the risk/benefit calculation is genuinely uncertain and the unknowns are real. This section documents these protocols honestly, including what we do not know, because people are running them regardless. Better information is safer than no information. None of this constitutes medical advice or recommendation.

Exp — 01
🔬 FrontierAnimal Data + Community N=1High Risk Profile
The Senolytic Clearance Stack
FOXO4-DRI · Dasatinib + Quercetin · Zombie Cell Elimination
Senescent "zombie" cells accumulate with age — they stop dividing but refuse to die, secreting inflammatory signals (SASP) that damage surrounding tissue. This protocol combines the peptide senolytic FOXO4-DRI with the small-molecule D+Q combination for mechanistic redundancy. Bryan Johnson documented follistatin gene therapy at the same clinic running early klotho trials. The senolytic community considers this the most targeted anti-ageing intervention currently accessible.
Stack — Compounds
FOXO4-DRI
1mg SubQ · 5 days on / 2 days off · 2-week course · 2–3x per year with 3–6 month rest. D-retro-inverso design makes it protease-resistant. Reconstitute in BAC water, swirl gently — can take 5–10 min to dissolve. Protect from light.
→ Entry page
Dasatinib + Quercetin (D+Q)
100mg Dasatinib + 1000mg Quercetin oral · 2 consecutive days per month. The established small-molecule senolytic combination. Run separately from FOXO4-DRI — allow 2-week washout between FOXO4-DRI course and D+Q days. Never combine simultaneously.
Epitalon (supportive)
5–10mg SubQ · 10-day course · twice yearly — aligns telomerase activation with senescent cell clearance window. Many community members add Epitalon to the off-cycle period.
→ Entry page
🔬
Bryan Johnson context: Johnson received follistatin gene therapy at GARM Clinic in Próspera, Honduras — the same location now running Klothea Bio's klotho mRNA trial. He also trialed Cerebrolysin IM daily for 3 months (no measurable effect on his biomarkers). Johnson discontinued growth hormone use. His protocol prioritises measurable biomarker change over anecdotal experience — a useful framing for evaluating any experimental compound.
Why People Run This
⚡ The Hypothesis
Senescent cell burden increases with age and is causally linked to multiple age-related diseases. FOXO4-DRI disrupts the FOXO4-p53 interaction that keeps senescent cells alive — freeing p53 to trigger apoptosis selectively in senescent cells (11.73-fold selectivity for senescent over normal cells in vitro). D+Q targets different survival pathways, creating redundancy. The 2017 Baar et al. Cell paper showed restored fur density, kidney function, and fitness in aged mice. The hypothesis: periodic clearance of zombie cells removes the chronic inflammatory burden that drives accelerated ageing.
⚠ What We Don't Know
Long-term safety in humans is completely uncharacterised. Tolerance develops with repeated cycles — first-cycle clearance 40–60%, dropping to 15–25% by third cycle at same dose. Whether rapid senescent cell clearance overwhelms phagocytic capacity is unknown. Stem cell exhaustion risk — senescent cells have some protective functions in wound healing. Off-target effects on non-senescent cells with transient FOXO4 elevation not characterised. Human dose translation from mouse data (5mg/kg) is complex — community 1mg protocol is a deliberate conservative choice.
🔬 Minimum Before Attempting
Age: Community consensus: FOXO4-DRI is unlikely to benefit healthy individuals under 40 with low senescent cell burden. Baseline blood work: CBC, CMP, inflammatory markers (CRP, IL-6), epigenetic age test (TruAge). Contraindications: Active infection, recent surgery, active cancer or cancer history, immunocompromised. Never combine: Multiple senolytics simultaneously — always separate with minimum 4-week washout. Monitoring: Inflammatory markers between courses — the signal of senescent cell clearance.
Exp — 02
🔬 FrontierMostly Animal DataCommunity Running
Mitochondrial Maximalist Stack
SS-31 · MOTS-c · Humanin · Klotho-Derived Peptides · The Full Mito Protocol
The complete mitochondrial peptide stack — combining the three major mitochondria-encoded peptides (SS-31, MOTS-c, Humanin) with klotho-derived peptides for the most comprehensive mitochondrial and longevity signalling protocol currently accessible. Each addresses a different aspect of mitochondrial decline. Klotho-derived peptides are the newest addition — 2025 mouse data showed 19.7% lifespan extension from soluble klotho administration.
Stack — Compounds
SS-31 (Elamipretide)
2–4mg SubQ · 5x/week · ongoing. The most evidence-backed in this stack — FDA-approved for Barth syndrome (as Bendavia). Targets mitochondrial inner membrane cristae structure. The anchor compound — start here and add others progressively.
→ Entry page
MOTS-c
5–10mg SubQ · 3–5x/week. Mitochondria-encoded, AMPK-activating, insulin-sensitising. Elevated in centenarians. Exercise-mimetic metabolic effects. Community running alongside SS-31 — overlapping but non-redundant mechanisms.
→ Entry page
Humanin
2–4mg SubQ or IM · 3x/week. Cytoprotective mitochondrial peptide — declines with age, neuroprotective, anti-apoptotic in healthy cells. Works upstream of MOTS-c in mitochondrial signalling cascade.
→ Entry page
Klotho-Derived Peptides
0.01–0.1mg SubQ · weekly · highly experimental. Soluble alpha-klotho fragments. The 2025 Molecular Therapy paper showed 15–20% lifespan extension in mice. No established human protocol — community extrapolating from animal data. Cost is significant.
→ Entry page
Why People Run This
⚡ The Hypothesis
Mitochondrial function declines predictably with age — reduced ATP production, increased ROS, impaired cristae structure, declined AMPK signalling. Each compound in this stack targets a different node of that decline. SS-31 addresses cristae structure and electron transport efficiency. MOTS-c restores AMPK-driven metabolic flexibility. Humanin provides cytoprotection and declines measurably with age. Klotho modulates IGF-1, Wnt, and FGF23 signalling with pleiotropic organ protection. The hypothesis: comprehensive mitochondrial support addresses multiple ageing mechanisms simultaneously rather than one node in isolation. MOTS-c's presence in centenarian blood at higher levels than age-matched controls is the most compelling human signal in this stack.
⚠ What We Don't Know
The combination of all four has no safety data. SS-31 has FDA approval context but at different indications. Klotho-derived peptide delivery is technically challenging — klotho is a large protein and peptide fragments may not fully replicate the native protein's effects. The 2025 mouse lifespan data is extraordinary but mouse-to-human translation for longevity interventions has historically been unreliable. MOTS-c's long-term effects on stem cell populations are not characterised. Whether synergy between these compounds is additive, synergistic, or antagonistic in combination is unknown.
🔬 Minimum Before Attempting
Start with SS-31 alone for 4 weeks before adding subsequent compounds — assess individual response before layering. Baseline: Epigenetic clock test, mitochondrial function markers if available, inflammatory panel, fasting glucose/insulin, full metabolic panel. Klotho-derived peptides specifically: Only add after 3+ months of SS-31/MOTS-c/Humanin combination — and only from verified research sources with confirmed sequencing. Annual reassessment: Repeat epigenetic clock — this is the most meaningful outcome measure for this stack.
Exp — 03
🔬 FrontierUnknown Risk ProfileAdvanced Nootropic Community
Cognitive Maximalist Stack
Dihexa · Cerebrolysin · NA Semax Amidate · Selank · Maximum BDNF & HGF Protocol
The frontier cognitive enhancement protocol — combining Dihexa (potentially 10 million times more potent than BDNF in activating HGF/c-Met signalling) with Cerebrolysin's multi-factor neurotrophic support and intranasal peptide modulation. Bryan Johnson trialed daily Cerebrolysin IM for 3 months. The nootropic community's most advanced stack — and the one with the least human safety data.
Stack — Compounds
Dihexa
10–20mg oral or 1–2mg intranasal · 2–3x/week. HGF/c-Met agonist — described as 10 million times more potent than BDNF in some assays for synaptogenesis stimulation. Oral bioavailability reasonable. Intranasal preferred by advanced users for CNS delivery. Cycle: 4 weeks on, 4 weeks off minimum.
→ Entry page
Cerebrolysin
5–10mL IM · daily for 10-day courses · 2–4x/year. Multiple neurotrophic factors (BDNF, NGF, CNTF). Bryan Johnson ran 5mL IM daily for 3 months — reported best subjective mental clarity he had experienced. No measurable biomarker effect on his panel. Requires verified cold-chain source (Ebewe Pharma, Austria).
→ Entry page
N-Acetyl Semax Amidate
200–400mcg intranasal · morning · 5/2 cycle. BDNF upregulation. Works through different mechanism from Dihexa (ACTH-derived vs HGF/c-Met). Widely considered safer — run this first as a baseline before adding Dihexa.
→ Entry page
Selank
250mcg intranasal · as needed. Anxiolytic companion — prevents overstimulation from Semax. Some community members report Semax-induced anxiety is amplified when Dihexa is also running.
→ Entry page
🔬
Bryan Johnson: Explicitly documented Cerebrolysin — 5mL IM daily for 3 months. Called it the best thing subjectively for mental clarity. His biomarker panel showed no measurable effect. This is an important data point: subjective cognitive enhancement may not produce measurable biochemical changes on standard panels. Johnson discontinued growth hormone use and reported trialing many peptides not publicly documented.
Why People Run This
⚡ The Hypothesis
Dihexa specifically: Most cognitive peptides upregulate BDNF or modulate receptor sensitivity. Dihexa works differently — it activates the HGF/c-Met pathway which directly promotes synaptogenesis (new synapse formation). This is not receptor sensitisation but structural CNS change. Improved spatial memory in aged rats at concentrations far below effective BDNF doses. Combined with Cerebrolysin's broad neurotrophic support, the hypothesis is that the two compounds address different aspects of cognitive decline — Dihexa drives new synaptic architecture; Cerebrolysin provides the neurotrophic growth factor environment to sustain it.
⚠ What We Don't Know
Dihexa has essentially no human safety data. The mechanism (promoting HGF/c-Met signalling) has theoretical cancer promotion concerns — c-Met is overexpressed in many cancers. Whether the doses used in cognitive enhancement contexts are sufficient to promote tumour growth is unknown. Off-cycle periods are non-negotiable. Cerebrolysin sourcing is the most common failure point — counterfeit preparations are common and inactive. Bryan Johnson's null biomarker result with verified Cerebrolysin is an important data point — subjective effects and measurable effects may diverge significantly.
🔬 Minimum Before Attempting
Sequence matters: Run NA Semax Amidate alone for 4+ weeks first. Add Selank to assess anxiety response. Only then add Cerebrolysin in a 10-day course. Dihexa is the final addition — after understanding your individual response to the above. Contraindications for Dihexa: Any personal or family history of cancer, active inflammatory conditions, pregnancy. Blood work: Full inflammatory panel, liver function (quarterly on Dihexa), cognitive benchmarking (Cambridge Brain Sciences or similar) at baseline and monthly.
Exp — 04
🔬 FrontierBryan Johnson ContextGene Therapy Adjacent
Follistatin + Klotho Longevity Combination
Recombinant Follistatin · Klotho-Derived Peptides · The Longevity Protein Stack
The two proteins most associated with lifespan extension in animal models, now available in peptide/recombinant form for self-experimentation. Bryan Johnson received follistatin gene therapy at GARM Clinic in Próspera — the same site now running Klothea Bio's Phase 1b klotho mRNA trial. Recombinant follistatin and klotho-derived peptides are accessible versions of what gene therapy delivers. The combination is described in the serious biohacking community as producing synergistic effects beyond either alone.
Stack — Compounds
Recombinant Follistatin (rFST)
50–100mcg SubQ · weekly or bi-weekly. Myostatin inhibitor — binds and neutralises myostatin (GDF-8) to release the brake on muscle growth. Gene therapy version costs ~$25,000 per treatment; recombinant peptide form is accessible but short half-life limits effect duration. Also inhibits activin A — anti-inflammatory implications.
→ Entry page
Klotho-Derived Peptides
0.01–0.1mg SubQ · weekly · highly experimental. Alpha-klotho declines from age 40. The 2025 Molecular Therapy study: soluble klotho extended mouse lifespan 19.7%, improved brain, muscle, and bone. A 2023 Nature Aging study: single low-dose klotho injection enhanced memory in aged primates. No established human peptide protocol — community extrapolating from animal and primate data.
→ Entry page
SS-31 (mitochondrial support)
2–4mg SubQ · 5x/week. Supportive — addresses the mitochondrial axis while Follistatin and Klotho address muscle/metabolic and organ protection respectively. Many serious longevity biohackers run all three simultaneously.
→ Entry page
🔬
Bryan Johnson explicitly received follistatin gene therapy in October 2023 at GARM Clinic in Próspera, Honduras — the same experimental biotech zone where Klothea Bio is now running Phase 1b klotho mRNA trials. This convergence of follistatin gene therapy and klotho research in one location reflects the serious longevity community's view that these two proteins represent the most compelling accessible longevity targets. Johnson's documented interest represents the upper end of what the biohacking community is pursuing — the peptide versions are more accessible versions of the same hypothesis.
Why People Run This
⚡ The Hypothesis
Follistatin: Myostatin is the body's natural brake on muscle growth. Follistatin inhibits myostatin — removing that brake. Beyond muscle: follistatin also inhibits activin, with anti-inflammatory and potentially anti-fibrotic implications. Gene therapy (as Bryan Johnson received) provides sustained expression; recombinant peptide requires more frequent administration but is far more accessible. Klotho: Alpha-klotho decline is one of the most consistent biomarkers of ageing. It modulates IGF-1 signalling (reducing cancer promotion while preserving anabolic benefits), protects kidneys, brain, cardiovascular system, and bone. The 2025 lifespan data in mice is the most compelling longevity peptide data published to date.
⚠ What We Don't Know
Recombinant follistatin half-life is short — weekly dosing may not maintain sustained myostatin inhibition. Whether the peptide form replicates gene therapy outcomes is unconfirmed. Klotho peptide delivery: Klotho is a large protein (~130 kDa). Derived peptide fragments may not replicate native protein activity across all signalling pathways. The primate and mouse lifespan data used full-length or soluble klotho — not peptide fragments. The interaction between follistatin-driven myostatin inhibition and klotho-driven IGF-1 modulation may be antagonistic in some pathways (myostatin inhibition increases muscle IGF-1 sensitivity; klotho reduces IGF-1 signalling). This theoretical interaction is undocumented in combination.
🔬 Minimum Before Attempting
Recombinant follistatin: Not for use with any active or recent cancer. Myostatin inhibition is contraindicated with hormonally sensitive cancers. Run blood work including IGF-1, full hormonal panel, and tumour markers at baseline. Klotho-derived peptides: No established safety threshold. Start with the lowest published dose and extend observation period before any escalation. Test kidney function at baseline — klotho is a kidney-produced protein and renal status is relevant. Both: Annual comprehensive cancer screening before and during use.
Exp — 05
🔬 FrontierAnimal Data OnlyEarly Community Experimentation
SLU-PP-332 Exercise Mimetic Protocol
ERRα/δ Agonist · Exercise in a Molecule · Endurance Without Training
SLU-PP-332 activates the ERR (oestrogen-related receptor) family — the transcription factors that drive the metabolic adaptations to endurance exercise. The 2023 Salk Institute study showed 70% improvement in running capacity in sedentary mice with no training. The community is calling it the most promising exercise mimetic compound ever identified. Zero human data. The community is running it anyway.
Stack — Compounds
SLU-PP-332
10–20mg oral · daily. ERRα/δ/γ agonist — activates the same transcriptional programme that endurance training activates. Oral bioavailability reasonable in animal models. Community extrapolating human dose from mouse data. Cycle: 8 weeks on, 4 weeks off as cautious starting approach.
→ Entry page
MOTS-c (synergistic addition)
5mg SubQ · 3x/week. AMPK activator — complementary mechanism to SLU-PP-332's ERR activation. Both increase mitochondrial biogenesis through different pathways. Many community members running both for additive metabolic effect.
→ Entry page
Berberine (mitigation)
500mg oral · 2–3x/day with meals. AMPK activator — provides evidence-based metabolic support alongside the experimental compounds. Also manages any glucose dysregulation. Used as the "insurance" compound in this stack.
Why People Run This
⚡ The Hypothesis
Endurance exercise activates ERR transcription factors which drive mitochondrial biogenesis, fatty acid oxidation, and metabolic flexibility. SLU-PP-332 activates these same transcription factors pharmacologically. The 2023 Salk Institute data: sedentary mice showed 70% improved running endurance and 12% reduced weight gain on high-fat diet without any exercise. The mechanism is not stimulant-based — it genuinely activates the cellular machinery of endurance adaptation. For populations with mobility limitations, chronic illness, or simply those wanting to amplify training adaptations, the potential is extraordinary. The community framing: AICAR was the first exercise mimetic (banned by WADA within a year of the 2008 endurance paper); SLU-PP-332 is considered mechanistically superior with broader ERR family activation.
⚠ What We Don't Know
Zero human safety or efficacy data exists. The ERR transcription factor family has complex roles in cancer biology — ERRα in particular is overexpressed in breast and ovarian cancers. Whether pharmacological ERR activation promotes cancer growth is entirely uncharacterised. Long-term metabolic effects of sustained ERR agonism are unknown. Whether the profound mouse endurance effects translate to humans is not established — exercise mimetics have historically translated poorly from rodents. WADA status: currently not listed, but this may change rapidly if human evidence emerges.
🔬 Minimum Before Attempting
Contraindications: Absolute contraindication with any hormone-sensitive cancer history (ERR family involvement). Women with BRCA mutations or family history of breast/ovarian cancer — do not use. Blood work: Full metabolic panel, fasting glucose, inflammatory markers at baseline. VO2 testing if available — the most objective outcome measure for an exercise mimetic. Cycling: Do not run continuously — the off-period is important given the unknown long-term ERR activation profile. WADA: Prohibited in competitive sport is likely a matter of when not if — check current list before competing.
Exp — 06
🔬 FrontierBryan Johnson AdjacentMaximum Complexity
Longevity Maximalist Protocol
Epitalon · FOXO4-DRI · SS-31 · MOTS-c · Humanin · Klotho · GHK-Cu · NAD+ · The Full Stack
The complete longevity peptide protocol — combining all established and frontier compounds for comprehensive hallmarks-of-ageing coverage. This is what the serious longevity community and Bryan Johnson-adjacent self-experimenters are running. Each compound addresses a different ageing mechanism. The full combination has no safety data as a complete stack. This is genuinely n=1 territory.
Stack — Compounds
Foundation Layer (run first, 3+ months)
SS-31 (daily) + GHK-Cu (topical daily + SubQ 3x/week) + NAD+/NMN oral (daily) + Collagen peptides (daily). These have the strongest evidence base. Start here before adding anything else.
Established Experimental Layer
Epitalon (10-day courses 2x/year) + MOTS-c (3x/week) + Humanin (3x/week) + Thymosin α-1 (twice weekly, 12-week courses 2x/year). Add these after 3 months on foundation layer.
Frontier Layer (experienced only)
FOXO4-DRI (2-week courses 2–3x/year, separate from everything else with washout) + Klotho-derived peptides (weekly, highly experimental) + Recombinant follistatin (bi-weekly). Only after 6+ months on the layers above.
Epigenetic Monitoring (non-negotiable)
TruAge or equivalent epigenetic clock at baseline, 6 months, 12 months. This is the only objective measure of whether the protocol is working. Bryan Johnson uses this as his primary longevity outcome metric.
🔬
Bryan Johnson's documented protocol includes: Follistatin gene therapy, daily Cerebrolysin trials, collagen peptides (20–30g daily), epigenetic age monitoring as primary outcome, multiple physician team oversight, and $2 million/year in total spend. The peptide stack above is the community-accessible version of the same underlying philosophy. Johnson's key contribution to this space is the insistence on measurement — running experimental protocols without biomarker tracking is self-experimentation without data, which is simply risk without information.
Why People Run This
⚡ The Hypothesis
The rationale is coverage of the major hallmarks of ageing simultaneously rather than addressing one mechanism in isolation: Telomere/telomerase (Epitalon) · Senescent cell burden (FOXO4-DRI) · Mitochondrial function (SS-31, MOTS-c, Humanin) · NAD+ decline (NMN/NR + 5-Amino-1MQ) · Tissue quality (GHK-Cu, Collagen) · Immune senescence (Thymosin α-1) · IGF-1/organ protection (Klotho-derived) · Myostatin braking (Follistatin). Bryan Johnson's Blueprint protocol addresses these same hallmarks through overlapping mechanisms — this stack is the peptide-focused version of that philosophy.
⚠ What We Don't Know
The combination of all compounds simultaneously has no safety data. Interactions between frontier compounds are completely unknown. FOXO4-DRI's senolytic activity plus Thymosin α-1's immune activation during the same window could theoretically create an inflammatory burden from rapid senescent cell clearance — the washout approach addresses this partially. Klotho's IGF-1 modulation may interact with follistatin's myostatin inhibition in complex ways. The practical complexity of running this correctly — with appropriate washouts, timing, and monitoring — is substantial. Most people attempting the full stack simultaneously are making the protocol worse, not better. The layered sequencing approach is not optional.
🔬 Minimum Before Attempting
Sequence is everything: Foundation → Established → Frontier. Never start at the frontier layer. Annual requirements: Epigenetic clock, telomere length, full comprehensive blood panel, cancer screening appropriate for age, DEXA bone density. Non-negotiables: Never run FOXO4-DRI within 2 weeks of D+Q or other senolytics. Never run during active infection, injury recovery, or surgery. The honest minimum: If you cannot afford the monitoring, you cannot afford this protocol. The compounds are the smaller cost — the testing is the real investment.
Clinic — 08
Age-Related Decline Replacement Protocol
Comprehensive 40+ Restoration · Hormonal · GH Axis · Mitochondrial · Vascular · Cognitive
From age 40, a predictable set of declines begins — testosterone, growth hormone, NAD+, GHK-Cu, IGF-1, and multiple other critical molecules fall measurably. This protocol addresses each decline systematically, replacing what the body progressively stops making with the closest available biological equivalents. Not anti-ageing fantasy — a structured response to documented, measurable decline.
Physician Supervised 40+ Protocol Risk: Moderate — monitoring essential
What Declines After 40 — And What We Replace It With
What Declines Rate of Decline Effect Replacement / Support
Testosterone ~1–2% per year from 35+ Libido ↓ · muscle mass ↓ · energy ↓ · cognitive clarity ↓ · bone density ↓ TRT (physician) + Gonadorelin to preserve HPG axis
Growth Hormone / IGF-1 ~14% per decade from 30+ Body composition ↓ · recovery ↓ · skin quality ↓ · sleep depth ↓ · muscle repair ↓ Sermorelin or Ipamorelin/CJC nightly + MK-677
NAD+ ~50% by midlife in many tissues Mitochondrial efficiency ↓ · DNA repair ↓ · sirtuin activity ↓ · energy metabolism ↓ NMN/NR oral daily + periodic IV/IM NAD+ infusion + 5-Amino-1MQ
GHK-Cu (Copper Peptide) Significant age-dependent decline Collagen synthesis ↓ · wound healing ↓ · skin quality ↓ · gene regulation decline GHK-Cu topical 1% daily + SubQ 0.5–1mg 3×/week
DHEA / DHEA-S Peaks at ~25, falls 50% by 50 Precursor to sex hormones ↓ · immune function ↓ · energy ↓ · mood ↓ DHEA 25–50mg oral (if blood test confirms low DHEA-S)
Mitochondrial Function Progressive from 40+ · accelerating Energy production ↓ · exercise capacity ↓ · cellular stress resilience ↓ SS-31 (elamipretide) + MOTS-c + Humanin cycling
Thymic function / T-cells Thymus involutes from 20s onward Immune surveillance ↓ · cancer risk ↑ · infection susceptibility ↑ · autoimmune risk ↑ Thymosin α-1 1.5mg twice weekly · 12-week courses 2×/year
Collagen Synthesis ~1% per year from 25+ Skin laxity ↑ · joint health ↓ · bone density ↓ · tendon/ligament fragility ↑ Collagen peptides 15–20g daily + Vitamin C 500mg
Melatonin Production Significant decline from 40+ Sleep architecture ↓ · circadian rhythm disruption · antioxidant capacity ↓ Epitalon (normalises pineal melatonin) + low-dose melatonin if needed
Klotho Protein Declines from ~40 · strongly age-associated Organ protection ↓ · cognitive decline ↑ · kidney function ↓ · cardiovascular risk ↑ Klotho-derived peptides (experimental) · exercise raises endogenous klotho
Foundation Layer — Start Here (Month 1–3)
Collagen Peptides + Vitamin C
♂ 15–20g oral daily + 500mg Vit C  ·  ♀ 10–15g oral daily + 500mg Vit C
The accessible non-negotiable foundation. Collagen synthesis declines 1% per year from 25 — no peptide protocol is complete without addressing this. Take together 30 min before exercise or consistently morning.
→ Entry page
NAD+ / NMN oral + periodic IV/IM
♂♀ 500–1000mg NMN oral daily + IV 500mg monthly or IM 250mg weekly
NAD+ declines ~50% by midlife. Oral NMN maintains baseline elevation. Monthly IV infusion or weekly IM injection provides deeper tissue restoration. Combine with 5-Amino-1MQ to block NAD+ catabolism simultaneously.
→ Entry page
GHK-Cu (Copper Peptide)
♂♀ Topical 1% cream daily + 0.5–1mg SubQ 3×/week
Endogenous GHK-Cu falls significantly with age. Topical for skin and scalp; SubQ for systemic gene regulation (activates 4,000+ genes). Women often see enhanced response due to oestrogen-collagen synergy.
→ Entry page
Sermorelin (GH axis restoration)
♂ 300mcg SubQ nightly  ·  ♀ 200mcg SubQ nightly — women need lower dose
Growth hormone declines ~14% per decade. Sermorelin restores natural pulsatile GH release rather than replacing it. Safer and more physiological than exogenous HGH. Monitor IGF-1 at 8 weeks — target mid-normal for age.
→ Entry page
Hormonal Layer — Add Month 3+ (Physician Required)
Testosterone Replacement (if confirmed low)
♂ 80–200mg testosterone cypionate SubQ weekly  ·  ♀ 5–20mg SubQ weekly
Only if blood test confirms below-normal levels — not for optimisation to supraphysiological. Always with Gonadorelin (men) to preserve HPG axis. Women: use free testosterone not total — SHBG critical. Physician supervised.
Gonadorelin (male TRT companion)
♂ 100–200mcg SubQ twice daily alongside testosterone
Maintains HPG axis pituitary connection during TRT. Prevents testicular atrophy. LH and FSH should remain measurable. If LH undetectable at 8 weeks — add Kisspeptin-10.
→ Entry page
MK-677 (GH axis — sleep depth)
♂ 10–25mg oral nightly  ·  ♀ 5–10mg oral nightly — women reach therapeutic dose lower
Add to Sermorelin for 24-hour GH elevation. Primary benefit: sleep quality (stage 4 slow-wave sleep — documented in RCTs). Monitor fasting glucose — MK-677 induces mild insulin resistance at higher doses. Add berberine 500mg 2×/day if glucose trends up.
→ Entry page
Thymosin α-1 (immune restoration)
♂♀ 1.5mg SubQ twice weekly · 12-week courses · twice yearly
Thymic involution from the 20s progressively impairs immune surveillance. Thymosin α-1 restores T-regulatory function and NK cell activity. Run spring and autumn — immune reset aligned with seasonal transitions. 35+ country approvals.
→ Entry page
Advanced Layer — Add Month 6+ (After Foundation + Hormonal Established)
SS-31 + MOTS-c
Mitochondrial function restoration. SS-31 (elamipretide) — FDA breakthrough for Barth syndrome. 2–4mg SubQ 5×/week. MOTS-c — 5mg SubQ 3×/week. Cycle 12 weeks on / 4 weeks off alternating.

♂♀ Same dose — no significant sex difference
Epitalon
Pineal gland restoration — telomerase activation, melatonin normalisation, circadian rhythm regulation. 5–10mg SubQ daily × 10 days. Twice yearly (January + September). Addresses the melatonin decline of ageing through pineal support rather than direct melatonin replacement.

♂♀ Same dose
5-Amino-1MQ
NNMT inhibitor — blocks the enzyme that wastes NAD+ precursors, raising intracellular NAD+ alongside NMN/NR supplementation. Complementary mechanisms: NMN adds supply; 5-Amino-1MQ reduces consumption. 50mg oral daily. Cycle 12 weeks on / 4 weeks off.

♀ Potentially enhanced thermogenic effect
⚠ Key Risks by Layer — With Mitigations
moderate TRT — haematocrit elevation — Monitor every 3 months. Must stay below 52% (men) / 48% (women). Therapeutic phlebotomy (blood donation) if elevated. Mitigation: weekly SubQ injections produce more stable levels and lower haematocrit vs fortnightly IM.
moderate MK-677 — insulin resistance — Fasting glucose elevation at 25mg/day is near-universal without mitigation. Mitigation: berberine 500mg 2–3×/day with meals. HbA1c quarterly. Dose at 10mg if glucose sensitive.
low Sermorelin — IGF-1 overshoot — Some individuals produce more IGF-1 than intended. Mitigation: blood test at 8 weeks. Target mid-normal for age — not upper limit. Reduce dose if above target.
low IV NAD+ — infusion rate reaction — Flushing, nausea, chest tightness if infused too rapidly. Mitigation: clinic-administered, slow rate (over 2–4 hours minimum). Rate control by the infusion nurse resolves this completely.
low Thymosin α-1 — immune activation response — Fatigue and mild flu-like symptoms after first 1–2 injections. Expected response. Mitigation: reduce to once weekly initially. Resolves within the first week of dosing.
serious Female TRT — voice deepening — The one irreversible androgenic side effect. Cannot be reversed after it occurs. Mitigation: start at absolute minimum (5mg/week), titrate 2.5mg every 4–6 weeks. Stop immediately at first sign of any voice change.
🔬 Comprehensive Blood Work Schedule — Age-Related Decline Protocol
Before Starting Comprehensive baseline
Full Hormonal Panel Once
  • Total + free testosterone
  • SHBG · oestradiol · LH + FSH
  • DHEA-S · prolactin · cortisol (morning)
  • Thyroid: TSH + free T3/T4
  • PSA (men 40+)
GH Axis Panel Once
  • IGF-1 — non-negotiable baseline
  • Fasting GH (optional)
  • Fasting glucose + HbA1c + fasting insulin
Longevity Biomarkers Baseline then annually
  • Epigenetic clock (TruAge or equivalent)
  • NAD+ blood level
  • hsCRP + IL-6 (inflammation)
  • Lipids: total, LDL, HDL, apoB
  • Full blood count + haematocrit
  • Liver + kidney function
  • Vitamin D · DHEA-S
During — First Year Quarterly minimum
Month 2 — GH Titration Month 2
  • IGF-1 — Sermorelin response check
  • Fasting glucose (MK-677 monitoring)
  • Haematocrit (if on TRT)
Month 3 Quarterly Panel Every 3 months
  • Total + free testosterone (trough if TRT)
  • Oestradiol · LH + FSH
  • Haematocrit — must stay below 52%♂ / 48%♀
  • Fasting glucose + HbA1c
  • IGF-1 · hsCRP
  • PSA (men 45+, every 6 months)
♀ Female-Specific Timing Days 8–12 of cycle
  • All hormone tests premenopausal
  • Free testosterone + SHBG
  • DHT if androgenic signs develop
Annual Comprehensive Once per year
Full Annual Panel Annually
  • Complete hormonal panel repeat
  • IGF-1 · full metabolic panel
  • Epigenetic clock repeat — primary outcome
  • NAD+ blood level
  • Comprehensive cancer screening (age-appropriate)
  • DEXA bone density (if on TRT 2+ years)
  • Cardiovascular risk assessment
  • Mammography ♀ (age-appropriate)
  • Thyroid full panel
Protocol Outcome Tracking Annually
  • Epigenetic age vs chronological age
  • Body composition (DEXA preferred)
  • Muscle mass and strength benchmarks
  • Cognitive function testing
  • Energy + sleep quality scores
♀ Female note: Women generally need lower doses across all GH-axis compounds (Sermorelin 200mcg vs 300mcg, MK-677 5–10mg vs 10–25mg). Free testosterone is more important than total testosterone for women. SHBG can mask adequate total testosterone by binding the active fraction. Premenopausal women: always test at consistent cycle phase. The epigenetic clock is the most objective annual outcome measure for the full protocol — compare to sex-matched reference ranges.
Section Four

Fitness Community Protocols

Strongman · Bodybuilding · Aesthetics · Endurance — with mitigation, blood work & male/female doses

Four distinct communities, four different goal hierarchies. Every protocol leads with the stack and the solutions — protective co-supplements, smart monitoring, and the practical steps that reduce risk for people who are going to run these protocols anyway. Framed as informed, intelligent use rather than a warning list.

Fitness — 01
Strongman / Powerlifting
Maximum Strength · Connective Tissue Resilience · Injury Prevention
Strongman and powerlifting place the most extreme mechanical load on connective tissue of any sport. The peptide strategy is dual: performance (HGH + IGF-1 LR3 + secretagogues) and continuous injury prevention (BPC-157 + TB-500). In this sport, the connective tissue stack is as important as the performance stack — one torn tendon erases what months of performance compounds build.
CommunityRisk: High — full monitoring required
Stack — Male / Female Doses
HGH (Somatropin)
♂ Male
2–3 IU/day SubQ
♀ Female
0.5–1 IU/day SubQ
Performance range. Connective tissue under extreme load demands GH-driven collagen synthesis. Both sexes: daily SubQ abdomen. Full IGF-1 monitoring non-negotiable.
→ Entry page
IGF-1 LR3
♂ Male
30–60mcg post-training · 4–6wk cycles
♀ Female
15–30mcg post-training · 4–6wk cycles
Post-training injection only. ALWAYS have 30–50g fast carbs ready — for both sexes. Maximum 4–6 week cycles with equal time off. Women report pronounced effects at half the male dose.
→ Entry page
Ipamorelin / CJC-1295
♂ Male
200/100mcg 2–3x daily
♀ Female
200/100mcg 2x daily
Foundation secretagogue — run continuously. Pre-sleep injection mandatory. Preserves pituitary function alongside exogenous HGH.
→ Entry page
BPC-157 (continuous)
♂ Male
500mcg SubQ daily near joint/tendon load sites
♀ Female
250–500mcg SubQ daily
Continuous year-round connective tissue protection — the most important protocol decision for strongman. Inject near highest-load sites (knees, hips, shoulders, lower back). Not cycled.
→ Entry page
TB-500
♂ Male
2mg SubQ twice weekly
♀ Female
1.5–2mg SubQ twice weekly
Systemic connective tissue support alongside BPC-157. Loading during competition build-up, maintenance through season.
→ Entry page
Protocol & Smart Approach

Dual-stack philosophy: Performance (HGH + IGF-1 LR3 + secretagogues) and injury prevention (BPC-157 + TB-500 continuous) run simultaneously. In strongman, one missed training block from a torn tendon costs more than any performance stack gains. Injury prevention is equally important as performance.

IGF-1 LR3 cycling: 4–6 week blocks timed to competition build-up phases. Never continuous. Equal off-time. HGH can run 8–12 week cycles with 4–6 weeks off.

GH gut prevention: Keep IGF-1 blood levels below 2× upper limit of normal. Test monthly. Reduce dose if approaching this threshold.

Competition timing: For tested athletes — stop IGF-1 LR3 at least 3–4 weeks before. Continue BPC-157 and TB-500 (not prohibited). Verify WADA list annually.

🛡️ Mitigation Layer — Protective Co-Supplements
🌿
TUDCA + NAC
TUDCA 500mg + NAC 600mg daily
Liver protection during HGH + IGF-1 LR3 cycles. TUDCA protects bile duct cells; NAC replenishes glutathione. Run throughout all heavy cycles.
🫐
Berberine
500mg 3x daily with meals
Essential insulin sensitiser — both HGH and IGF-1 LR3 elevate glucose. Non-negotiable from day 1. Switch to Metformin if glucose trends above 100 mg/dL despite berberine.
🌱
Collagen Peptides + Vitamin C
20g collagen + 1g Vitamin C 30–60 min pre-training
Delivers collagen peptides to tendons during loading — the timing with mechanical stimulus has RCT support. The best dietary intervention for connective tissue resilience.
🦴
Vitamin D3 + K2 + Magnesium
D3 5000 IU + K2 200mcg + Mg 400mg daily
Bone mineralisation under extreme load. Vitamin D deficiency significantly increases stress fracture risk. K2 directs calcium to bone not arteries. Magnesium for muscle function and sleep quality.
🐟
Omega-3
5–6g EPA+DHA daily
Maximum anti-inflammatory dose. Reduces joint inflammation from mechanical load, cardiovascular protection from HGH glucose effects, reduces joint pain during heavy training blocks.
🍬
Fast carbs — always accessible
30–50g glucose tabs in gym bag + car + home
IGF-1 LR3 hypoglycaemia emergency protocol. Non-negotiable regardless of sex. Have these ready every single training session.
🔬 Blood Work — Baseline · During · Post
Baseline
IGF-1 — essential
Fasting glucose + HbA1c + insulin
Full liver panel
Full blood count + haematocrit
Kidney function
Lipid panel
Thyroid
Full hormonal panel
PSA (men 40+)
Abdominal girth measurement
During Protocol
IGF-1 monthly — keep below 2× ULN
Fasting glucose monthly
Liver enzymes every 6 weeks
Blood glucose 1h post IGF-1 LR3 dose
Haematocrit quarterly
Abdominal girth monthly (GH gut early warning)
Post-Cycle
IGF-1 4 weeks post-cycle
Full liver panel 8 weeks post
Fasting glucose 6 weeks post
Annual cardiac assessment for elite competitors
♀ Female note: Women use 30–50% lower HGH and IGF-1 LR3 doses. Female IGF-1 target: 120–230 ng/mL (vs male performance range 180–300 ng/mL). Women are more sensitive to fluid retention and glucose effects — monitor more frequently in first 8 weeks. Connective tissue peptides: similar doses to men, often better relative response.
WADA 2025HGHIGF-1 LR3BPC-157 (not listed)TB-500 (not listed)Ipamorelin/CJC (not listed)
Fitness — 02
Bodybuilding — Mass & Competition Prep
Maximum Muscle Mass · Phase-Based Approach · GH Gut Prevention
Two distinct phases with different compound selections. Mass: maximum anabolic signalling through HGH + IGF-1 LR3 + MGF + secretagogues. Prep: shift to conditioning — drop IGF-1 LR3, add AOD-9604 for fat targeting, maintain GH for skin quality. GH gut is real, documented, and preventable — the solution is informed dosing and monthly IGF-1 monitoring.
CommunityRisk: High — comprehensive monitoring
Stack — Male / Female Doses
HGH
♂ Male
Mass: 2–4 IU/day · Prep: 2 IU maintain
♀ Female
Mass: 0.5–1.5 IU · Prep: 0.5–1 IU
Mass phase upper limit 4 IU — above this GH gut risk rises substantially. Prep phase: maintain HGH for skin tightening and conditioning. Women: 0.5–1.5 IU mass, 0.5–1 IU prep.
→ Entry page
IGF-1 LR3 (mass phase only)
♂ Male
40–80mcg post-training · strict 4–6wk cycles
♀ Female
20–40mcg post-training · 4–6wk cycles
Mass phase only — stop or significantly reduce during prep. Strict 4–6 week maximum then equal off-time. This cycling is the primary GH gut prevention strategy.
→ Entry page
MGF / PEG-MGF
♂ Male
PEG-MGF 200mcg SubQ twice weekly
♀ Female
PEG-MGF 100–150mcg SubQ twice weekly
Satellite cell activation — non-redundant with IGF-1 LR3. Women respond very well at lower doses. Mass phase compound — stop during prep phase.
→ Entry page
Ipamorelin / CJC-1295
♂ Male
200/100mcg 3x daily
♀ Female
200/100mcg 2x daily
Foundation through both phases. Pre-sleep most important injection. Preserves pituitary pulsatility during exogenous HGH.
→ Entry page
AOD-9604 (prep phase)
♂ Male
300mcg fasted morning
♀ Female
200–250mcg fasted morning
Competition prep replacement for IGF-1 LR3. GH fragment targeting fat mobilisation without IGF-1 elevation. The phase switch: stop IGF-1 LR3, add AOD-9604.
→ Entry page
BPC-157 (continuous)
♂ Male
500mcg SubQ daily
♀ Female
250–500mcg daily
Year-round connective tissue protection. Bodybuilding joint wear accumulates — BPC-157 continuously is injury insurance through both phases.
→ Entry page
Protocol & Smart Approach

GH gut — what it is and how to prevent it: Visceral organ and GI smooth muscle hypertrophy from chronic supraphysiological IGF-1. Prevention: keep IGF-1 blood levels below 2× upper limit of normal. Cycle IGF-1 LR3 strictly — 4–6 weeks maximum with equal off-time. Take breaks from HGH above 2 IU. If abdominal distension appears — reduce dose immediately. This is the most visible long-term consequence of aggressive GH protocols and the one most easily prevented by sensible monitoring.

Phase approach: Mass phase (months 1–4+): HGH + IGF-1 LR3 + PEG-MGF + Ipamorelin/CJC + BPC-157. Prep phase: Stop IGF-1 LR3 and PEG-MGF. Maintain HGH. Add AOD-9604 fasted morning. Continue Ipamorelin/CJC and BPC-157.

Female bodybuilding: GH gut risk exists in women too — same monitoring applies. Women achieve competitive results at lower doses. Do not escalate to male doses assuming more is needed. Hormonal panel essential — prep cycles can significantly disrupt menstrual function affecting bone health long-term.

🛡️ Mitigation Layer — Protective Co-Supplements
🌿
TUDCA + NAC
TUDCA 500mg + NAC 600mg daily
The bodybuilder liver stack. Long peptide cycles (often alongside AAS) create hepatic stress. TUDCA protects bile duct cells; NAC replenishes glutathione. Run throughout all heavy cycles without exception.
🫐
Berberine
500mg 3x daily with meals
Non-negotiable insulin sensitiser at bodybuilding peptide doses. Both HGH and IGF-1 LR3 elevate glucose. From day 1 of any HGH + IGF-1 LR3 cycle. Switch to Metformin if insufficient.
❤️
CoQ10 / Ubiquinol
200–400mg daily with fat-containing meal
Cardiac mitochondrial protection. Bodybuilding-level training and peptide stacks place significant cardiovascular demand. Ubiquinol (reduced form) has better absorption. Critical if also using AAS.
🐟
Omega-3
6g EPA+DHA daily
Maximum dose — anti-inflammatory, cardiovascular protection, insulin sensitisation, joint protection. The single most important supplement for long-term bodybuilding health. Both phases.
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Taurine + Vitamin D3/K2
Taurine 3–5g + D3 10000 IU + K2 200mcg daily
Taurine: cardiac muscle protection, anti-arrhythmic, reduces cramping. D3/K2: bone density and arterial calcium management during elevated calcium turnover in aggressive protocols.
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Regular blood donation if haematocrit elevates
Every 8–12 weeks if haematocrit approaching 52%
If AAS are used alongside peptides, haematocrit management becomes critical. Blood donation is the most effective intervention. Even peptide-only users should monitor haematocrit quarterly.
🔬 Blood Work — Baseline · During · Post
Baseline
IGF-1 — non-negotiable
Full liver panel
Full blood count + haematocrit
Fasting glucose + HbA1c + insulin
Lipid panel (apoB ideal)
Kidney function
ECG + cardiac panel
Abdominal girth
Full hormonal panel
Thyroid
During Protocol
IGF-1 monthly — keep below 2× ULN (the GH gut prevention metric)
Fasting glucose monthly
Liver enzymes every 6 weeks
Abdominal girth monthly
Haematocrit quarterly
Blood pressure monthly
Lipids quarterly
Annual cardiac echo at high-level
Post-Cycle
IGF-1 4 weeks post
Full liver panel 8 weeks post
Fasting glucose 6 weeks post
Annual cardiac assessment
Full hormonal recovery panel 12 weeks post
♀ Female note: Women: 30–50% lower doses across the board. GH gut monitoring applies equally — women are not immune. Hormonal panel must include oestradiol — aggressive prep can cause amenorrhoea affecting bone density. Baseline DEXA bone density recommended for serious female competitors before first full prep cycle.
WADA 2025HGHIGF-1 LR3MGF/PEG-MGFBPC-157 (not listed)AOD-9604 (not listed)Ipamorelin/CJC (not listed)
Fitness — 03
Aesthetics / Physique Community
Lean Muscle · Low Body Fat · Skin Quality · Sustainable Approach
The most accessible fitness protocol — moderate doses, emphasis on consistency, and the addition of skin-quality peptides that matter more for aesthetics than raw mass. Where most community users actually sit. Excellent results without the risk profile of competitive bodybuilding.
CommunityRisk: Low–Moderate
Stack — Male / Female Doses
Ipamorelin / CJC-1295
♂ Male
200/100mcg 2–3x daily · 8–12wk cycles
♀ Female
200/100mcg 2x daily · 8–12wk cycles
The safest and most sustainable entry point. Highly selective — no cortisol or prolactin elevation. Pre-sleep injection most important. Both sexes tolerate identically.
→ Entry page
MK-677
♂ Male
15–25mg nightly at bedtime
♀ Female
10–15mg nightly at bedtime
Women: 10mg provides excellent sleep and body composition results without the hunger and water retention of 25mg. Start low and assess before escalating. Oral convenience.
→ Entry page
Low-dose HGH (optional)
♂ Male
1–2 IU/day SubQ
♀ Female
0.2–0.5 IU/day SubQ
Optional upgrade — women achieve meaningful aesthetics and skin results at 0.2–0.5 IU. Men: 1–2 IU is the aesthetics-appropriate range. Above 2 IU enters bodybuilding risk territory.
→ Entry page
GHK-Cu
♂ Male
Topical 1% daily + 0.5–1mg SubQ 3x/week
♀ Female
Topical 1–2% daily + 0.5mg SubQ 3x/week
Skin quality is a primary aesthetics goal. Women often respond more strongly to topical GHK-Cu. SubQ for systemic gene activation. Both sexes: topical daily is the foundation.
→ Entry page
BPC-157 (as needed)
♂ Male
250–500mcg SubQ at first sign of overuse
♀ Female
250mcg SubQ as needed
Aesthetics athletes train frequently — injury interrupts progress more than any compound compensates for. Use at first sign of overuse injury or during heavy training phases.
→ Entry page
Protocol & Smart Approach

Why this works: The secretagogue + MK-677 combination provides the hormonal environment for body composition change without the acute risks of IGF-1 LR3 or high-dose HGH. GHK-Cu is often overlooked by performance-focused communities but is central to the aesthetics outcome — skin quality, muscle definition, overall appearance.

The aesthetics timeline: Month 1: sleep improvement from MK-677 (often within first week). Month 2–3: visible body composition shift. Month 4–6: GHK-Cu skin quality improvement becomes visible. Month 6+: cumulative improvement.

Female aesthetics: Women consistently achieve excellent results at these conservative doses. MK-677: some women align with menstrual cycle, pausing during luteal phase (days 15–28) to manage water retention amplified by progesterone. Do not assume male doses are required.

🛡️ Mitigation Layer — Protective Co-Supplements
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Berberine (at 25mg MK-677)
500mg 2–3x daily with meals
MK-677 at 25mg elevates fasting glucose meaningfully. Berberine from day 1 at this dose. Less critical at 10–15mg but still beneficial for insulin sensitivity.
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Collagen + Vitamin C
15g collagen + 1g Vitamin C daily
Skin quality and joint resilience — both central to aesthetics goals. Synergistic with GHK-Cu collagen synthesis. Pre-workout timing for joint benefit.
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Omega-3
3g EPA+DHA daily
Anti-inflammatory, skin lipid barrier improvement (synergistic with GHK-Cu), cardiovascular support. The baseline supplement for any training protocol.
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Magnesium Glycinate + Zinc
Magnesium 300mg + Zinc 25mg nightly
Sleep quality (synergistic with MK-677), testosterone support, recovery. The most underrated supplement combination for aesthetics athletes.
🔬 Blood Work — Baseline · During · Post
Baseline
Fasting glucose + HbA1c (MK-677 baseline)
IGF-1 (if adding HGH)
Full blood count
Metabolic panel
Skin photography (baseline comparison)
During Protocol
Fasting glucose monthly at 25mg MK-677
HbA1c quarterly
Body composition photos every 4 weeks
Waist circumference every 2 weeks
Post-Cycle
Fasting glucose 4 weeks post-MK-677
IGF-1 4 weeks post (if HGH layer)
Body composition vs baseline
♀ Female note: Women achieve excellent aesthetics results at consistently lower doses. Female-specific tip: cycle MK-677 to align with menstrual cycle — pause during luteal phase (days 15–28) to avoid progesterone-amplified water retention. GHK-Cu: women typically report more dramatic facial skin improvement than men due to the oestrogen-collagen hormonal synergy.
Fitness — 04
Endurance — Marathon · Cycling · Triathlon
Mitochondrial Efficiency · Overuse Injury Prevention · Recovery · WADA Compliance
Endurance athletes have the most unique peptide use profile — less focused on anabolism, more on mitochondrial efficiency, recovery from high training volume, and chronic overuse injury management. WADA compliance is a real concern. The good news: the most useful compounds for endurance performance are largely not currently prohibited.
CommunityRisk: Low — mostly non-prohibited
Stack — Male / Female Doses
BPC-157
♂ Male
250–500mcg SubQ or oral · daily or 5x/week
♀ Female
250mcg SubQ or oral · daily or 5x/week
Primary endurance peptide — overuse injury prevention and management. Plantar fasciitis, stress fractures, IT band, runner's knee. Oral acceptable for gut and systemic effects. Not WADA prohibited.
→ Entry page
TB-500
♂ Male
2mg SubQ twice weekly during heavy training blocks
♀ Female
1.5mg SubQ twice weekly
Systemic tissue repair during peak training phases. Run during 4–6 week high-volume blocks. Systemic — site injection less critical than BPC-157.
→ Entry page
MOTS-c
♂ Male
5–10mg SubQ 3–5x/week
♀ Female
5mg SubQ 3–5x/week
Mitochondrial AMPK activation — the most relevant endurance-specific peptide mechanism. Improves insulin sensitivity and metabolic efficiency. Not currently WADA prohibited. Women: excellent response at 5mg.
→ Entry page
SS-31 (Elamipretide)
♂ Male
2–4mg SubQ daily or 5x/week
♀ Female
2mg SubQ daily or 5x/week
Mitochondrial inner membrane protection — reduces oxidative damage from extreme training load. FDA approved for Barth syndrome. Protects mitochondria from cumulative endurance stress. Not WADA prohibited.
→ Entry page
NAD+ / NMN
♂ Male
500–1000mg NMN oral daily
♀ Female
500mg NMN oral daily
NAD+ replenishment — endurance training depletes NAD+ significantly. NMN has the best evidence for exercise performance application. Not prohibited.
→ Entry page
Protocol & Smart Approach

WADA compliance: BPC-157, TB-500, MOTS-c, SS-31, and NMN are not currently on the WADA Prohibited List. AICAR is prohibited (added 2009). HGH is prohibited. This protocol is designed to stay within permitted compounds for competitive athletes — verify the current WADA list annually as it updates.

Mitochondrial focus: MOTS-c + SS-31 + NMN address the three key mitochondrial failure points in endurance athletes: NAD+ depletion (NMN), mitochondrial ROS damage (SS-31), and metabolic efficiency (MOTS-c). Together they target the mitochondrial degradation that limits endurance performance and recovery.

Injury timing: BPC-157 + TB-500 most valuable during and immediately after peak training blocks. Start 4 weeks into a heavy block and run through to 2 weeks post-race.

Female athletes — iron monitoring is the most important single intervention: Iron deficiency is the most common performance-limiting condition in female endurance athletes and is frequently missed. Ferritin below 30 ng/mL impairs endurance performance even without clinical anaemia. Target ferritin above 50 ng/mL.

🛡️ Mitigation Layer — Protective Co-Supplements
🩸
Iron bisglycinate + Vitamin C (if ferritin deficient)
Iron bisglycinate 25–50mg + Vitamin C 500mg · away from caffeine by 2 hours
Most common performance-limiting deficiency in endurance athletes — especially premenopausal females. Iron bisglycinate has the best GI tolerability. Vitamin C doubles absorption. Non-negotiable if ferritin below 50 ng/mL.
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Electrolytes — sodium, potassium, magnesium
High-quality electrolyte supplement daily + intra-workout for sessions over 90 min
MOTS-c and NMN metabolic effects increase electrolyte demand. Endurance depletes all electrolytes — magnesium deficiency is universal in endurance athletes and impairs mitochondrial function directly.
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CoQ10 / Ubiquinol
Ubiquinol 200mg daily with fat-containing meal
Mitochondrial electron transport chain support — directly synergistic with SS-31. Ubiquinol (reduced form) better absorbed than CoQ10. Most impactful in athletes over 35 as endogenous production declines.
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Omega-3 (anti-inflammatory focus)
3g EPA + 1g DHA daily
Reduces inflammatory load from high training volume. EPA is the primary anti-inflammatory omega-3. Reduces post-race inflammatory markers and supports the injury prevention that BPC-157 addresses pharmacologically.
🔬 Blood Work — Baseline · During · Post
Baseline
Ferritin + full iron panel (haemoglobin, haematocrit, transferrin)
Full blood count (anaemia screen)
Inflammatory markers: hsCRP, IL-6
Morning cortisol (overtraining baseline)
RBC magnesium (preferred over serum)
Vitamin D
Metabolic panel
Thyroid (fatigue differential)
During Protocol
Ferritin every 8 weeks (endurance depletes iron rapidly)
Morning cortisol at peak training (overtraining syndrome marker)
hsCRP monthly
HRV and resting HR daily tracking
Pain and injury scores weekly
Post-Cycle
Full iron panel 4 weeks post major race
Cortisol + DHEA-S (HPA axis recovery)
Full blood count 4 weeks post season
Ferritin at start of each new training block
♀ Female note: Female endurance athletes: ferritin monitoring is non-negotiable. Target ferritin above 50 ng/mL (above 30 ng/mL minimum for performance). Premenopausal women may need iron supplementation continuously during high training volume — this is the most important single intervention for female endurance performance. MOTS-c dosing similar to men (5mg); BPC-157/TB-500 at lower end of range initially.
WADA 2025BPC-157 (not listed)TB-500 (not listed)MOTS-c (not listed)SS-31 (not listed)NMN (not listed)AICAR (prohibited 2009)HGH (prohibited)